1) Diabetic micro-angiopathy
糖尿病性微血管并发症
3) Diabetic cardiovascular complications
糖尿病慢性血管并发症
4) Diabetic vascular complication
糖尿病血管并发症
1.
Effect of coixan on expression of NO and iNOS mRNA of aorta in rats of experimental diabetic vascular complication;
薏苡仁多糖对糖尿病血管并发症大鼠NO及主动脉iNOS基因表达的影响
2.
Diabetic vascular complications are the main cause of cripples and death of the diabetic patients,and the hereditary factor might play the vital role in the morbidity process of complications.
糖尿病血管并发症是糖尿病患者主要的致残、致死原因,遗传因素可能在并发症的发病过程中发挥着重要的作用。
3.
Methods The in vitro cell-injuring model of diabetic vascular complication was used to observe the cyto-morphology changes, the cell growth vitalis and the contents of nitrogen monoxidum(NO), endothelin-1(ET-1), tissue-type plasminogen activator(tPA) and its inhibitor(PAI) in the conditioned medium after respectively adding three dose ZGE a.
方法采用2型糖尿病血管并发症细胞研究模型,观察不同剂量左归降糖灵及达美康作用后细胞形态学、细胞生长活力及条件培养液中NO、ET-1、tPA、PAI-1含量的变化。
5) Macrovascular Diseases
糖尿病大血管并发症
1.
The Research of the Association of the Level of Serum BH4 and Macrovascular Diseases induced by diabetes mellitus;
血浆BH4水平与2型糖尿病大血管并发症的相关性研究
补充资料：糖尿病非酮症性高渗性昏迷
糖尿病非酮症性高渗性昏迷
diabetic hyperosmolar nonketotic coma
糖尿病患者由于感染、尿毒症、电解质紊乱、甲亢、尿崩症等原因使血糖升高引起渗透性利尿，细胞内失水，特别是脑细胞可引起昏迷和死亡。特点起病慢，常被诱发本病的疾病或伴随症状所掩盖。早期呈原有糖尿病症状加重，嗜睡，逐渐进入昏迷状态。血糖＞28～54mmol/L，血钠＞145mmol/L，血浆渗透压＞310mmol/L，血、尿酮体无明显增高，无酸中毒。用等渗液或低于血浆渗透压40mmol/L的高渗液治疗，予以小剂量胰岛素，防止因血糖降低使血浆渗透压下降过快引起脑水肿。
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参考词条