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1)  audiogenic seizures
听源性惊厥
1.
We examined the effects of recurrent audiogenic seizures (AGSs) on BDNF protein expression in the brain of P77PMC rats using immunohistochemistry technique.
采用免疫组织化学方法观察了反复听源性惊厥 (AGS)对遗传性癫易感大鼠P77PMC脑内脑源性神经营养因子 (BDNF)蛋白表达的影响。
2)  audiogenic seizure
听源性惊厥
1.
The effects of block anesthetic lidocaine or convulsantia isoniazide of intrabrain injection on the audiogenic seizure were observed in P77-PMC inbred strain rats.
在P77-PMC品系大鼠上分别观察了阻滞麻醉剂利多卡因和致惊厥剂异烟肼脑内给药对听源性惊厥(AS)发作的影响。
3)  audiogenic kindling
听源性惊厥点燃
1.
To identify the neurochemical mechanisms of the audiogenic kindling in the forebrain structures, the expression of c fos gene, a marker of neuronal activity, and the level of brain derived neurotrophic factor (BDNF) in the neocortex were studied after single and kindled audiogenic seizure with immunocytochemistry method.
为探讨前脑结构参与听源性惊厥点燃过程的神经化学机制 ,本研究以 c-fos基因表达作为神经元功能活动的标志 ,采用免疫细胞化学方法 ,对新皮质内参与听源性惊厥点燃过程的神经元的分布状况进行了观察 ,并对相关区域内脑源性神经营养因子表达水平的变化进行了分析。
4)  febrile seizures
热性惊厥
1.
Effects of febrile seizures on emotion, behavior, learning and memory in rats;
热性惊厥对大鼠情感行为及学习记忆的影响
2.
The study of behavior problem in children with a history of febrile seizures
热性惊厥影响患儿行为的研究
3.
Objective To explore the clinical characteristics of 4 pedigrees of absence epilepsy with febrile seizures plus(FS+)in China.
目的探讨失神发作伴热性惊厥附加症(FS+)的临床特点。
5)  febrile seizure
热性惊厥
1.
Gene mutations involved in febrile seizures;
热性惊厥相关基因突变研究进展
2.
Sequencing GABRG2 gene in 2 Chinese families with generalized epilepsy with febrile seizures plus;
全身性癫癎伴热性惊厥附加症2个家系致病基因GABRG2测序分析
3.
Sequencing analysis of GABRA1 gene in linkage mapping generablized epilepsy with febrile seizures;
全身性癫痫伴热性惊厥附加症家系GABRA1基因测序分析
6)  febrile convulsion
热性惊厥
1.
Electroencephalographic representation and consequence of infant febrile convulsion;
小儿热性惊厥的脑电图表现及转归
2.
The Relationship between Serum Trace Elements and Children Febrile Convulsion;
小儿热性惊厥与血微量元素的关系
3.
Analysis of EEG in 200 patients with febrile convulsion;
200例小儿热性惊厥的脑电图分析
补充资料:良性特发性新生儿惊厥


良性特发性新生儿惊厥
benign idiopathic neonatal convulsions

男略多于女,起病于生后1~7天,最多在第5天,起病前正常。发病原因不明。发作形式以阵挛为主,多为限局性,也可有呼吸暂停发作,但没有强直发作。发作频,可有癫痫持续状态,持续数日。神经系统无异常体征。预后常良好,多数自动缓解。本症的诊断主要靠排除法。
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