1) Ca 2+ preconditioning
缺钙预处理
2) Calcium preconditioning
钙预处理
1.
Method: 32 rats were randomly divided into 4 groups: Ischemia/Reperfusion group (I/R group), Calcium preconditioning group (CPC group), Chelerythine (a PKC inhibitor) added into CPC group (CLT group) and 5 hydroxydecanoate (5 HD, a mitochondrial K ATP channel inhibitor) added into CPC group (5 HD group).
目的 :研究钙预处理是否对缺血再灌注心肌有保护作用 ,其作用是否通过蛋白激酶C(ProteinKinaseC ,PKC)及线粒体ATP依赖性钾通道 (KATP)起作用。
3) Ischemic preconditioning
缺血预处理
1.
Role of P_(38)MAPK/iNOS signal-transduction-cascades in the delay protection of renal ischemic preconditioning;
P_(38)MAPK/iNOS信号通路对肾脏缺血预处理的延迟保护效应
2.
Influence of 3-nitropropionic acid on expression of caspase-3 in brain tissue after ischemic preconditioning;
3-硝基丙酸对缺血预处理后脑组织caspase-3表达的影响
3.
Mechanism Study of Neuroprotective Effect of Ischemic Preconditioning;
缺血预处理脑保护机制研究
4) hypoxic preconditioning
缺氧预处理
1.
Protective effects of hypoxic preconditioning on the hypoxic/reoxygenation injury of neonatal rats cardiomyocytes;
缺氧预处理对乳鼠心肌细胞缺氧/复氧损伤的保护作用
2.
Objective To study the protective effects of hypoxic preconditioning (HPC) on myocardial contusion.
目的观察缺氧预处理(HPC)对大鼠钝性心肌挫伤的保护作用,并进一步探讨其机制。
3.
Objective To investigate observe the effects of asphyxial hypoxic preconditioning on neurological symptoms,expression of Hypoxia-inducible factor-1α(HIF-1α),content of nitric oxide(NO) and nitric oxide synthase(NOS) in brain tissue after intracranial hemorrhage(ICH) in rats.
目的观察停通气缺氧预处理(HPC)对脑出血(ICH)模型大鼠神经行为学评分、血肿周围组织中缺氧诱导因子-1α(HIF-1α)的表达和一氧化氮(NO)含量、一氧化氮合酶(NOS)活性的影响。
5) ischemic precondition
缺血预处理
1.
Protective effect of donator liver ischemic preconditioning on posttransplant liver graft function in Chinese miniature pigs;
缺血预处理对小型猪肝移植的影响
2.
Effect of Ischemic Precondition on Myocardial ATP and Na~+-K~+-ATPase Activity;
缺血预处理对大鼠心肌三磷酸腺苷含量及钠-钾-三磷酸腺苷酶活性的影响
3.
Expressions of AKT and JNK in mitochondria after cerebral ischemic precondition in rats
AKT及JNK激酶在大鼠脑缺血预处理后线粒体中的表达
6) ischemic preconditioning
缺氧预处理
1.
ObjectiveTo investigate the mechanism and the protective effect of heat shock protein 27 (HSP_27) on rat cardiomyocytes when ischemic preconditioning performed.
目的探讨大鼠心肌细胞缺氧预处理中热休克蛋白27(HSP27)的保护作用及其机制。
2.
OBJECTIVE ①To investigate the role of P38MAPK, iNOS, COX-2 and mitochondrial K+ATP channel, and explore the association among them in the late phase of ischemic preconditioning (IPC) in neonatal rat cardiomyocytes.
目的：（1）在大鼠心肌细胞缺氧预处理（ischemic preconditioning，IPC）晚期保护作用模型上，探讨丝裂素活化蛋白激酶P38（P38MAPK）、诱生型一氧化氮合成酶（iNOS）、环氧化酶-2（COX-2）和线粒体K+ATP通道的作用及相互关系。
补充资料：缺钙
缺钙
血清钙离子低于人体正常值水平。常见于小儿，如维生素D缺乏性佝偻病早期、甲状旁腺功能减退、碱中毒等。当血清钙低于7.5mg／dl或离子钙低于4mg／dl时，神经肌肉兴奋性增高而引起惊厥或局部肌肉抽搐，如婴儿手足搐搦症，由于喉部肌肉痉挛，甚至可造成窒息死亡。此外，严重缺钙可导致心肌收缩力减弱，甚至引起心律失常等。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条