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1)  Coranary artery ischemic preconditioning
冠状动脉缺血预适应
2)  coronary ischemia
冠状动脉缺血
3)  off-pump CABG
缺血性冠状动脉硬化病
4)  ischemia preconditioning
缺血预适应
1.
The combination therapy of anti-myocardial ischemia has advantages of not only augmenting the therapy and reducing dose of some drugs, but also enhancing the myocardial ischemia preconditioning, reducing the product which is of damage to the heart tissue and the body, preventing the ischemic arrhythmias, and augmenting the rebuilding of the heart tissue and vessel tissue.
抗心肌缺血联合用药不但可减少药物剂量,降低不良反应,而且还能加强或诱导心肌缺血预适应,降低心肌有害产物,抗心律失常,加强受损心肌或血管的修复,从而提高对心肌缺血的治疗效果。
2.
Objective To study the regular pattern of heat shock protein(HSP70) produced after heat shock,myocardial ischemia or ischemia preconditioning in heart and liver of rat.
目的 探讨热处理、心肌缺血及缺血预适应后大鼠心脏、肝脏热休克蛋白 (HSP70 )的产生规律。
3.
Now there are more studies about ischemia preconditioning (IP)against ischemia injury.
缺血再灌注损伤导致的急性肾功能衰竭(Acute renal failure,ARF)发生率很高,最近有关缺血预适应(Ischemia Preconditioning,IP)预防缺血再灌注损伤的研究日益增多。
5)  Preconditioning [英][,pri:kən'diʃən]  [美][,prikən'dɪʃən]
缺血预适应
1.
Preconditioning reduces endothelin content in coronary sinus during ischemia reperfusion;
缺血预适应减少缺血再灌注冠状窦血浆内皮素的含量
2.
To observe whether there were some protective effects on these injuries which subjected to ischemia preconditioning (IPC) and postconditioning 2.
以机体抗氧化能力为指标,研究在体大鼠心肌缺血/再灌注对肺和脑组织的影响,观察缺血预适应和缺血后适应的改善作用。
6)  ischemic preconditioning
缺血预适应
1.
Early effects of ischemic preconditioning on expressions of IKKβ,NF-κB,TNF-α mRNA and changes in rat kidney allograft tissue;
缺血预适应对大鼠肾移植早期组织学及IKKβ、NF-κB、TNF-αmRNA表达的影响
2.
Effect of ischemic preconditioning on left ventricular remodeling in patients with acute myocardial infarction;
心肌缺血预适应对急性心肌梗死左室重构的影响
3.
Effect of noninvasive limb ischemic preconditioning on myocardium matris metalloproteinases induced by ischemia/reperfusion in rats;
无创性肢体缺血预适应对大鼠缺血再灌注损伤心肌基质金属蛋白酶的影响
补充资料:冠状动脉

  
  冠状动脉
  coronary artery

  供应心脏血液的动脉。分为左、右两支,分别称左冠状动脉和右冠状动脉。左冠状动脉起于主动脉左窦,随即分为前室间支和旋支,前者供应左心室前壁、右心室前壁一小部分及室间隔前2/3区域;后者供应左室侧壁,左室后壁及左心房。右冠状动脉起于主动脉右窦,沿途发出的分支分布于右心房、窦房结、房室结、右心室、室间隔后1/3及左心室后壁的一部分。如因病理变化而管腔缩小或阻塞,可导致心肌供血不全而发生心绞痛、心肌梗死和房室传导阻滞等。
  
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