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1)  prostatic cancer (CaP)
前列腺癌(CaP)
2)  Prostatic cancer
前列腺癌
1.
Detection of prostate stem cell antigen and serum prostate specific antigen of patients with prostatic cancer;
前列腺癌患者癌组织前列腺干细胞抗原、血清前列腺特异抗原检测
2.
Changes of Androgen Receptor Expression in Hormonal Dependent Prostatic Cancer and Hormonal Independent Prostatic Cancer;
雄激素依赖性和非依赖性前列腺癌雄激素受体表达水平的变化
3.
Value of prostate specific antigen in early diagnosis of prostatic cancer;
前列腺特异性抗原对前列腺癌早期诊断的价值
3)  prostate cancer
前列腺癌
1.
Improved synthesis of 17-(5′-isoxazolyl) androst-4,16-dien-3-one,a potential agent for the treatment of prostate cancer;
新的前列腺癌治疗剂17-( 5′-异噁唑基 )雄甾-4,16-二烯-3-酮合成方法的改进(英文)
2.
Establishment and application of transplantable TRAMP-C2 prostate cancer mice model;
TRAMP-C2可移植前列腺癌动物模型的建立及初步应用
3.
The Differential and Diagnostic Value for Prostate Cancer of the Serum Free PSA and Total PSA and Their Ratio;
血清游离PSA与总PSA及其比值对前列腺癌的鉴别诊断价值
4)  prostate carcinoma
前列腺癌
1.
Morphological expression of glutathione S-transferase P1 in prostate carcinoma;
谷光甘肽S-转移酶在前列腺癌中的形态学表达
2.
The relationship between neuroendocrine differentiated prostate carcinoma and drug-resistance to endocrine therapy;
前列腺癌神经内分泌化与内分泌治疗耐药的关系
3.
Toxic effects of CD-TK double suicidal gene system against prostate carcinoma cells;
CD-TK双自杀基因系统对前列腺癌细胞的杀伤作用
5)  prostatic carcinoma
前列腺癌
1.
Relationship between prostatic carcinoma label matter and diet formation;
前列腺癌标志物与年龄及膳食结构关系
2.
Diagnostic value on prostatic carcinoma by transrectal color Doppler flow imaging;
经直肠彩色多普勒超声对前列腺癌的诊断研究
3.
The expression of p53 and VEGF in human prostatic carcinoma and their clinical significances;
p53和VEGF在前列腺癌组织中的表达及临床意义
6)  prostate adenocarcinoma
前列腺癌
1.
Methods The expression of PSA was analysed by using ABC immunohistochemical staining in Prostate adenocarcinomas.
目的 探讨血清和组织中前列腺特异性抗原 (PSA )的表达水平 ,与前列腺癌 (Pca)组织学分级的相关性。
2.
Background: Prostate adenocarcinoma is one of the major malignancies of the genitourinary system.
背景和目的:前列腺腺癌(以下简称前列腺癌)是泌尿生殖系统的主要恶性肿瘤之一。
补充资料:CAP
分子式:
CAS号:

性质:结合在操纵子上启动子区域的蛋白质。它和cAMP结合于靠近RNA聚合酶结合位点。CAP和启动子的反应有利于RNA聚合酶—启动子复合物的形成。该复合物起正调控作用,使转录得以进行。添加葡萄糖会降低cAMP水平,并使CAP不表现活性,从而降低转录水平。

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