1) Steroid-induced ischemic necrosis
激素性缺血性坏死
2) steroid-induced osteonecrosis of the femoral head
激素性股骨头缺血坏死
1.
Recently, the pathogenesis of steroid-induced osteonecrosis of the femoral head has been determinated in many theories of decentralization.
目前对激素性股骨头坏死机制的研究已经提出许多分散的理论,但均未能完全阐明激素性股骨头缺血坏死的发病机理,至今仍有许多质疑。
3) SANFH
激素性股骨头缺血性坏死
1.
Objective: The purpose of this study is to clarify the role of organic element (Collagen、Proteoglycan、Fat ) in SANFH(Steroid-induced avascular necrosis of the femoral head)and the changes of organic element with the Bone metabolism of SANFH.
目的:测定激素性股骨头缺血性坏死患者股骨头下松质骨中有机物胶原蛋白、蛋白聚糖、脂肪含量与正常对照组中有机物含量的水平的变化,探讨有机物质胶原蛋白、蛋白聚糖、脂肪在激素性股骨头缺血性坏死骨代谢中的含量变化以及其可能发生的作用。
2.
Through the literature retrospection of steraid-induced avascularnecrosis of femoral head(SANFH) both in TCM and western medicine in recent years, comment the theoretic of this disease in TCM and modern medicine.
论文通过回顾近年来中西医对激素性股骨头缺血性坏死研究的有关文献，客观的评述了中医及现代医学对该病的理论认识，并综述了文献对本病治疗方法的相关研究，旨在探讨在本病的发病机理认识以及治疗方法选择上找到一些中西医结合点的可能性，并为本病的中西医结合治疗提供一些思路。
4) steroid-induced avascular necrosis of femoral head (SANFH)
激素性股骨头缺血性坏死(SANFH)
5) Ischemic necrosis
缺血性坏死
1.
Biomechanical study on predicting the collapse of ischemic necrosis of the femoral head;
股骨头缺血性坏死塌陷预测的生物力学研究
2.
Biomechanical study on the treatment of ischemic necrosis of the femoral head using memorial metal flexible cage in adult;
记忆合金网球支架治疗成人股骨头缺血性坏死的生物力学研究
3.
Role of hawkins sign in the diagnosis of ischemic necrosis of talus;
霍金斯征在诊断距骨缺血性坏死中的价值
6) Avascular necrosis
缺血性坏死
1.
Experiment of early magnetic resonance imaging findings of corticosteroid induced avascular necrosis of the femoral head;
皮质类固醇致股骨头缺血性坏死的磁共振成像早期改变的实验研究
2.
Analysis of interventional therapeutic effect on avascular necrosis of femoral head;
股骨头缺血性坏死介入治疗
3.
cases with avascular necrosis of femoral head(ANFH) are diagnosed and staged with X ray,SPECT,MRI based on image evaluation standard.
对 5 1例缺血性坏死股骨头 (ANFH)进行 X线、SPECT、MRI图像检查 ,根据早、中、晚期图像评价标准对其分别进行分期。
补充资料：急性出血性坏死性肠炎
急性出血性坏死性肠炎
是与产气荚膜芽胞杆菌感染有联系的一种急性炎症性肠炎。主要病理改变为肠壁小动脉内类纤维蛋白沉着、栓塞而致小肠出血坏死。临床上以腹痛、便血、呕吐、腹胀和发热为主要表现，严重时常出现休克、肠麻痹或并发肠坏死，肠穿孔和腹膜炎。本病以非手术疗法为主，加强全身支持疗法，纠正水电解质失常，解除中毒症状，积极防止中毒性休克和其它并发症。必要时才考虑手术治疗。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条