A study on arsenic trioxide inhibiting in vitro growth of human colon cancer cells;

Expression of FAK in colon cancer cell lines and it s effect on cancer cell migration;

Apoptosis of colon cancer cell induced by Arsenic trioxide and its relation to cell cycle;

Establishment of gefitinib-resistant human colon carcinoma cell line HT-29/ZD and its drug resistant mechanism;

Anti-tumor effects of pIRApoptinHNIL18 on colon carcinoma cell HCT-116;

After renewal of culture medium,the KCs were cultured for another 24 hrs,together with mice colon carcinoma cells,CT 26 at the effect or/target ratio of 10:1.

Aim To investigate whether leptin can affect the injury induced by 5-fluorouracil (5-FU) on human colon cancer cell line HT-29 and examine the mechanisms involved.

Methods:After colon cancer SW480 cell were treated with TRAP, MTT assay, and Western blot, electrophoretic mobilityshift assay were used to detect the suppression rate ,cyclooxygenase-2 protein, nuclear factor κBactivity.

Methods After colon cancer LoVo cell were treated with docetaxel,the supression rate,telomerase activity,apoptosis and cell cycle of colon cancer Lovo cell were studied by MTT assay,TRAP-microtiter plate hybridization assay,agarose gel electrophoresis and terminal uridine deoxynucleotidyl nick end labeling(TUNEL),and flow cytometry,respectively.

Objective: To inhibit VEGF expression by RNA interference in vitro, and observe the apoptosis of Colon cancer cell when VEGF was knocked down, so that RNA interference could be used in clinical therapy.