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1)  advanced-glycation end-product cross-links breaker
晚期糖基化交联产物裂解剂
2)  advanced glycation end products
晚期糖基化终产物
1.
Effects of advanced glycation end products on proliferation and apotosis of human renal tubular epithelial cells;
晚期糖基化终产物对人肾小管上皮细胞增殖和凋亡的影响
2.
Effect of advanced glycation end products on the oxidative damage of PC12 cells induced by amyloid beta protein 25-35;
晚期糖基化终产物在β样淀粉蛋白25-35诱导PC12细胞氧化损伤中的作用
3.
Effects of Rosiglitazone on Advanced Glycation End Products in Atherosclerosis Rats;
罗格列酮对动脉粥样硬化大鼠血清晚期糖基化终产物的影响及可能机制
3)  Advanced glycation end-products
晚期糖基化终末产物
1.
Estrogen Attenuates the Inhibition of Nitric Oxide Synthase Type 3 Induced by Advanced Glycation End-Products
雌激素减弱晚期糖基化终末产物对内皮型一氧化氮合酶的抑制作用
2.
Objective To analyze the effects of different purification modalities in eliminating advanced glycation end-products(AGEs) in patients with uremia,and to evaluate their related clinical value.
目的通过不同透析方式对晚期糖基化终末产物(AGEs)清除作用进行比较,探讨能更好清除中分子毒素的透析方式,指导临床治疗。
3.
Objective To investigate the changes of serum advanced glycation end-products(AGEs) level and the relationship among serum AGEs,liver function and fibrosis markers in patients with 2 type of diabetic mellitus(2DM).
目的探讨2型糖尿病患者血清晚期糖基化终末产物(AGEs)水平变化及其与肝功能的关系。
4)  advanced glycation end product
晚期糖基化终产物
1.
Advanced glycation end products and diabetic erectile dysfunction;
晚期糖基化终产物与糖尿病性勃起功能障碍
2.
Advanced glycation end products inhibit production of nitric oxide by human endothelial cells through activation of the p38 signal pathway;
晚期糖基化终产物通过p38信号通路抑制内皮细胞合成一氧化氮的研究
3.
Objective:To investigate the role of advanced glycation end products(AGEs) and their receptors(RAGE) in the pathogenesis of diabetic mellitus erectile dysfunction(DMED) and the effects of AGEs and RAGE on the activity of endothelin-1(ET-1) in rat cavernosum.
目的:通过观察大鼠阴茎海绵体组织中晚期糖基化终产物(AGEs)及其受体(RAGE)的变化对内皮素1(ET-1)活性的影响,探讨AGEs在糖尿病性勃起功能障碍(DMED)发生发展中的作用。
5)  Advanced glycation end products
晚期糖基化终末产物
1.
Effects of Advanced Glycation End Products on Prostacyclin and Prostaglandin E_2 Expression in Culture Endothelial Cell and Its Mechanism;
晚期糖基化终末产物对内皮细胞前列腺素合成的影响及可能机制
2.
Methods The expression of advanced glycation end products ( AGEs) in the diabetic and non-diabetic skin were detected by immunohistochemistry assay.
方法对糖尿病患者皮肤组织中晚期糖基化终末产物(AGEs)的表达进行免疫组化测定;体外将FB的ECM糖基化处理,观察FB细胞的形态变化和糖基化基质对其黏附、增殖能力的影响,以非糖尿病皮肤组织作为对照。
3.
This paper is to study the effect of N-acetylcysteine on advanced glycation end products (AGEs) induced VCAM-1 expression in mouse cardiac microvascular endothelial cells.
用流式细胞仪测定晚期糖基化终末产物(advanced glycation end products,AGEs)刺激及N-乙酰半胱氨酸(NAC)干预前后小鼠心脏微血管内皮细胞血管细胞黏附分子1(VCAM-1)的表达,同时用放免法测定培养上清中TNF—α水平。
6)  AGEs [英][eɪdʒ]  [美][edʒ]
晚期糖基化终末产物
1.
In in-vivo experiments,the expression of AGEs contents was determined by fluorography and EGF levels by ELSA in both group rat skins.
方法STZ(streptozotocin,链脲佐菌素)诱导正常SD大鼠,成模后8周(晚期糖尿病大鼠模型),荧光自显影技术检测皮肤组织中晚期糖基化终末产物(advanced glycation end-products,AGEs)的沉积;ELSA法检测表皮层中EGF(epidermal growth factor表皮生长因子)含量;提取大鼠KCs,用二甲基噻唑二苯基四唑溴盐(MTT)法测定细胞增殖速度;检测细胞周期;观察KCs核内NF-κB活性。
2.
Advanced glycation endproducts(AGEs)are chemical modifications of proteins by carbohydrates including those metabolic intermediates formed during the Maillard reaction.
晚期糖基化终末产物(Advanced glycation endproducts,AGEs)是一类经由糖,包括通过Maillard反应形成的代谢中间产物化学修饰的蛋白。
3.
Objective To explore the clinical features of early diabetic cardiomyopathy, the early diagnostic value and significance of UCG, serous concentration of AGEs and P-ⅢNP in this disease.
记录年龄、性别、糖尿病病程,分别检测三组的体重指数(BMI)、腰围臀围比值(WHR)、血压、空腹血糖(FBG)、餐后2小时血糖(2HPG)、糖基化血红蛋白(HbA1c)、血脂全套分析、血尿酸(UA)、尿微量白蛋白(MUA)、血晚期糖基化终末产物(AGEs)和Ⅲ型前胶原氨基末端肽(PⅢNP)。
补充资料:有机过氧化物交联剂
分子式:
CAS号:

性质:是一种塑料用典型交联剂。过氧化物RCOOCR中的—OO—键的键能很小,受光或热的作用易分解产生自由基,该自由基可引发交联反应。其特点是可以和与硫黄等交联剂所不能交联的饱和聚合物进行交联,形成—C—C—交联键。可交联绝大多数聚合物,交联物的压缩永久变形小,无污染性,耐热性好。但在空气存在下交联困难,易受其他助剂的影响,交联反应中的残余物会产生令人不快的臭味,与硫黄硫化相比其交联物的机械性能略低。常用的有氢过氧化物、二烷基过氧化物、二酰基过氧化物、过氧酯、酮过氧化物。

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