1) pulmonary microvascular permeability
肺微血管通透性
1.
Methods:To establish an acute lung injury model in rats in vivo by intravenous lipopolysaccharide(LPS) challenge, observe the changes of parameters of partial pressure of oxygen,the bronchoalveolar lavage fluid (BALF), the pulmonary microvascular permeability index(PMPI), and the histological appearance of lung under microscope and electromicroscope in LPS group, LPS+PTX gro.
目的 :探讨己酮可可碱 (PTX)对内毒素 (L PS)诱导的急性肺损伤肺微血管通透性的影响。
2) Pulmonary vascular permeability
肺血管通透性
1.
Changes in Pulmonary vascular permeability index and pulmonary function during orthotopic liver transplantation
原位肝移植围术期肺血管通透性和肺功能的变化
3) Microvascular permeability
微血管通透性
1.
Changes of VEGF expression and pulmonary microvascular permeability by thrombin injection in rats;
凝血酶致大鼠肺微血管通透性及血浆VEGF含量改变的实验研究
2.
Objective: To study the effect of satragaloside Ⅳ on the microvascular permeability induced by histamine in pial microvessels.
目的 :观察黄芪皂苷甲对组胺引起的脑软膜微血管通透性增加的抑制作用。
3.
Objective To observe the changes of the microvascular permeability after blunt chest trauma (BCT), endotoxemia and their combined injury in rats.
目的 观察大鼠胸部撞击伤、内毒素血症及其复合损伤时肺组织微血管通透性变化规律。
4) Alveolar capillary permeability
肺泡毛细血管通透性
5) pulmonary capillary membrane permeability
肺毛细血管膜通透性
1.
Will the pulmonary capillary membrane permeability change with obstructive jaundice? Will the hyperbilirubinemia play a more important role in ARDS? There is little answer about it.
乌司他丁通过调节炎症因子的释放,抑制氧自由基的产生,改善循环状态,减少因阻黄引起的肺毛细血管膜通透性的增加,起到保护肺组织细胞的作用。
6) PVPI
肺血管通透性指数
1.
With PiCCO monitoring,Extravascular Lung Water(EVLW),Global End-Diastolic Volume(GEDV),Intrathoracic Blood Volume(ITBV) and Pulmonary Blood Volume(PBV) were collected and then Pulmonary Vascular Permeability Index (PVPI) was calculated and analyzed.
目的通过脉波指示剂连续心排血量(PiCCO)监护仪,评估肺血管通透性指数(PVPI)对于急性呼吸窘迫综合征(ARDS)诊断的临床价值。
补充资料：血管通透性
血管通透性
vascular permeability
毛细血管使蛋白、细胞和组织液等透过血管壁的性能。这些物质可以通过血管壁进入血流，也可从血流透过血管壁进入组织。正是由于血管的通透性才使得血液与组织间不断地进行物质交换，将营养物质运输到各组织间隙，把组织细胞代谢的产物又透过血管壁进入血液运送到排泄器官排出体外。毛细血管壁的正常结构改变时，血管通透性增高，使血管内液体过多渗出进入组织间隙。常由于细菌、毒素、某些化学因子、机械性因素、烧伤、冻伤、缺氧以及某些变态反应等原因使连接内皮细胞的粘合质及基膜受损；同时也损伤了血管周围组织，引起组胺及活性肽等的释放；受损局部的组织也因缺血、缺氧而使酸度增加。这些因素均可导致血管通透性增加，其结果使大量的血浆蛋白及液体渗出，形成水肿。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条