1) mitochondrial permeability transition pore
线粒体PT孔
1.
The method of literature was applied to discussing the relationship between mitochondrial permeability transition pores and mitochondrial transmembrane potential Δ m, apoptosis factors as well as Bcl—2 family, and probing into the possible mechanism of mitochondrial permeability transition pores associating themselves with apoptosis induced by exercise and its potential value in exercise training.
采用文献资料法,论述线粒体PT孔的重要调控因素及其与线粒体跨膜电位Δψm、凋亡因子、Bcl-2家族的关系;探讨线粒体PT孔参与运动诱导凋亡的可能机制和在运动训练中的潜在价值。
2.
Effects of mitochondrial permeability transition pore specific inhibitor cyclosporine A (CsA) and highly expressed Bcl 2 on the apoptosis of HL 60 cells induced by EGTA were studied.
研究线粒体PT孔专一抑制剂环孢菌素A(CsA)和Bcl 2高表达对EGTA诱导HL 6 0细胞凋亡的影响。
2) Mitochondrial permeability transition pore
线粒体转换孔
3) mitochondrial permeability transition pore
线粒体通透性转换孔
1.
The role and mechanism of mitochondrial permeability transition pore in ischemic postconditioning;
线粒体通透性转换孔在缺血后处理中的作用和机制
2.
The opening state of mitochondrial permeability transition pore determines the mitochondrial membrane permeability, which is crucial to regulate apoptosis: cytochrome c is released from mitochondria to cytoplasm, activates caspases via combining Apaf-1 and ATP, initiating caspases cascade, .
线粒体膜通透性转换孔的开放状态决定了线粒体膜的通透性,在调节细胞凋亡中起了非常关键的作用:细胞色素c从线粒体释放到胞浆中,与凋亡蛋白激活因子-1和ATP结合可活化caspases,启动caspases的级联反应,引起底物蛋白水解、凋亡小体形成等,诱发细胞凋亡;Bcl-2和Bax是细胞凋亡过程中两个重要的调控基因,Bcl-2的表达增强会抑制细胞凋亡,而Bax的过度表达则会促进细胞凋亡;诱导细胞凋亡时发生的线粒体Ca~(2+)摄取既诱发线粒体通透性转换孔的开放也介导线粒体发生钙诱导的钙释放,胞浆高浓度的钙能激活各种蛋白激酶而诱导细胞凋亡的发生。
3.
Aim: To investigate the role of mitochondrial permeability transition pore(MPTP) in the cardioprotection by remote preconditioning(RPC).
目的:观察线粒体通透性转换孔在远距预处理心肌保护中的作用。
4) mitochondrial permeability transition pore
线粒体膜通透性转换孔
1.
The effect of copper on the mitochondrial permeability transition pore was significant,resulting in swelling of mitochondria to some degree which was manifested by decr.
结果显示,铜对线粒体膜通透性转换孔有较显著的影响,造成线粒体不同程度的肿胀,表现为线粒体悬液在540 nm处的吸光值下降,且铜浓度越高,吸光值下降越明显,在孵育6 min后3种浓度的吸光值下降百分比差异显著(P<0。
5) Mitochondrial permeability transition pore
线粒体渗透转换孔
6) Mitochondrial permeability transition pore
线粒体通透转换孔
1.
Mitochondrial permeability transition pore and myocardial ischemia-reperfusion injury;
线粒体通透转换孔与心肌缺血-再灌注损伤
2.
ATP-sensitive potassium channel and mitochondrial permeability transition pore involve in cardioprotection of polydatin
ATP敏感性钾通道和线粒体通透转换孔参与白藜芦醇苷的心肌保护作用(英文)
3.
Objectives:The purpose of this study is with randomized controlled methods,by observing the differents among the four groups of SD rats of clean grade: cytochrome C oxidase activity and mitochondrial permeability transition pore\' s activity to explore the relations between the mitochondrial oxidative damage and osteoporosis.
结果:线粒体通透转换孔30min/0minOD比值比较显示,空白对照组和尼尔雌醇组与模型组有明显差异(P<0。
补充资料：第二孔(继发孔)型缺损
第二孔(继发孔)型缺损
ostium secondary defect
是心房间隔在形成上发生障碍，一般缺损较大，多在卵圆孔附近称第二孔(继发孔)型缺损。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条