1) intractable glomerular nephritis
难治性肾小球肾炎
1.
To discuss the thinking of and experience in the treatment of intractable glomerular nephritis with the therapy of Chinese medicine.
目的 :介绍笔者运用中医药方法治疗难治性肾小球肾炎的经验 ,探讨中医药治疗此类疾病的思路和方法。
2) mesangial glomerulonephritis (MESGN)
肾小球膜性肾小球肾炎
3) chronic glomerulonephritis
慢性肾小球肾炎
1.
Clinical investigation of the correlation between TNF-α and proteinuria in patients with chronic glomerulonephritis treated with angiotensin Ⅱ blockers;
血管紧张素Ⅱ阻断剂治疗慢性肾小球肾炎时TNF-α和蛋白尿的相关性研究
2.
Effect of Yishen capsule on serum vascular endothelial growth factor and cell immunity in patients with chronic glomerulonephritis;
益肾胶囊对慢性肾小球肾炎患者血清血管内皮生长因子及细胞免疫的影响
3.
Analysis of therapeutic effect on treatment of chronic glomerulonephritis with sodium ferulate;
阿魏酸钠治疗慢性肾小球肾炎54例疗效分析
4) acute glomerulonephritis
急性肾小球肾炎
1.
Expression and prognosis prediction of MCP-1 in children with acute glomerulonephritis;
急性肾小球肾炎患儿单核细胞趋化蛋白-1的表达及意义
2.
Methods Paraffin-embedded renal biopsy tissue sections from 28 children with primary nephrotic syndrome(PNS), 18 children with acute glomerulonephritis(AGN), 20 children with Henoch-Schoenlein purpura nephritis(HSPN) and 22 children with isolated hematuria(IHU) were examined by immunohistochemical staining with anti-FGFR 4 monoclonal antibody.
方法采用免疫组化法观察FGFR4在原发性肾病综合征(28例)及急性肾小球肾炎(18例)、紫癜性肾炎(20例),单纯性血尿(22例)患儿及正常肾组织(5例)的表达。
3.
Objective:To investigate the role of nitric oxide(NO)and tumor necrosis factor α(TNF α)in the pathogenesis of acute glomerulonephritis(AGN).
目的 :探讨血清一氧化氮 (NO)和肿瘤坏死因子 α(TNF α)在小儿急性肾小球肾炎 (AGN)中的作用及其相互关系。
5) Membranous glomerulonephritis
膜性肾小球肾炎
1.
Influence of Shenkang decoction on experimental membranous glomerulonephritis in rabbits;
肾康饮对家兔实验性膜性肾小球肾炎的影响
6) acute glomerular nephritis
急性肾小球肾炎
1.
Objective: We studied the role of erythrocyte phospholipid peroxidation in acute glomerular nephritis (AGN).
目的:探讨急性肾小球肾炎（AGN）与磷脂过氧化的关系。
补充资料：膜性增殖性肾小球肾炎
膜性增殖性肾小球肾炎
membrano?proliferative glomerulonephritis
光镜下可见肾小球系膜细胞增生、系膜基质增加和扩展以及毛细血管壁增厚。电镜观察可分为3种亚型。Ⅰ型：增生的系膜细胞和基质伸入到基膜与内皮细胞之间，使毛细血管壁似乎分裂成双层而增厚，毛细血管腔被压而变窄；Ⅱ型：肾小球膜内出现电子致密沉积物，又称致密沉积物病；Ⅲ型：沉积物不规则地出现在毛细血管壁内。临床上本型多见于肾炎性肾病及慢性肾炎，血清C3持续明显降低，又称低补体血症性肾炎。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条