1) ischemic preconditioning
预缺血
1.
Rapid ischemic preconditioning protects rabbits against ischemic neuronal damage following global cerebral ischemia;
快速预缺血处理对兔脑再缺血的保护作用(英文)
2.
The effects of KATP channel opener cromaklim (Cro) and ischemic preconditioning(IP) on hemodynamics and myocardial infarct size were examined in both urethaneand sodium pentobarbital anesthetized rabbit models of myocardial ischemia-reperfusionto determine whether the KATP channel was involved in the cardioprotection provided byIP.
在氨基甲酸乙酯和成巴比妥钠两种不同麻醉的家兔心肌缺血－再灌注（IR）模型上，观察了ATP敏感性钾通道（KATP通道）开放剂cromaklim（Cro）和预缺血（IP）对血流动力学和心肌梗塞范围的影响，旨在阐明KATP通道是否参与IP对IR心肌的保护机制。
3.
Objective To investigate the effect of ischemic preconditioning on chaperone hsp70 expression and protein aggregation in the CA1 neurons of rats,and to further explore its potential neuroprotective mechanism.
目的研究预缺血对蛋白伴侣hsp70表达和蛋白聚集物形成的影响,探讨其可能的脑保护机制。
2) cerebral ischemic preconditioning
脑预缺血
1.
The adenosine (ADO) mechanism is an important explaining for the protective effects of cerebral ischemic preconditioning (CIP) on neurons.
腺苷机制是解释脑预缺血(cerebral ischemic preconditioning，CIP)神经保护作用的一个重要学说，即CIP引起脑细胞合成和释放腺苷(adenosine，ADO)增加，后者与其特异性受体结合发挥对神经元的保护作用。
2.
It has been illustrated that cerebral ischemic preconditioning (CIP) leads to increases in synthesis and release of adenosine.
目的:研究表明,腺苷在脑缺血耐受(brain ischemic tolerance, BIT)的形成中发挥重要作用,即脑预缺血(cerebral ischemic preconditioning, CIP)引起脑细胞合成和释放腺苷增加,后者与特异性受体结合发挥对神经元的保护作用,使之耐受损伤性缺血。
3) ischemic/hypoxic preconditioning
缺血/缺氧预适应
1.
Brain ischemic/hypoxic preconditioning induced alteration of genomic and proteomic expression;
脑缺血/缺氧预适应引起的基因、蛋白质表达谱的变化
4) ischemic/hypoxia preconditioning
缺血/缺氧预处理
1.
The model was used to analyze the effect of ischemic/hypoxia preconditioning(IPC/HPC) on angiogenesis of EMs.
目的建立子宫内膜异位症的鸡胚模型,分析缺血/缺氧预处理对子宫内膜异位症血管生成的影响。
5) ischaemic/hypoxic preconditioning
缺血\缺氧预处理
6) ischemia preconditioning
缺血预适应
1.
The combination therapy of anti-myocardial ischemia has advantages of not only augmenting the therapy and reducing dose of some drugs, but also enhancing the myocardial ischemia preconditioning, reducing the product which is of damage to the heart tissue and the body, preventing the ischemic arrhythmias, and augmenting the rebuilding of the heart tissue and vessel tissue.
抗心肌缺血联合用药不但可减少药物剂量,降低不良反应,而且还能加强或诱导心肌缺血预适应,降低心肌有害产物,抗心律失常,加强受损心肌或血管的修复,从而提高对心肌缺血的治疗效果。
2.
Objective To study the regular pattern of heat shock protein(HSP70) produced after heat shock,myocardial ischemia or ischemia preconditioning in heart and liver of rat.
目的 探讨热处理、心肌缺血及缺血预适应后大鼠心脏、肝脏热休克蛋白 (HSP70 )的产生规律。
3.
Now there are more studies about ischemia preconditioning (IP)against ischemia injury.
缺血再灌注损伤导致的急性肾功能衰竭(Acute renal failure,ARF)发生率很高,最近有关缺血预适应(Ischemia Preconditioning,IP)预防缺血再灌注损伤的研究日益增多。
补充资料：短暂性脑缺血发作
短暂性脑缺血发作
transient ischemic attack,TIA
急性脑血管病之一。指一时性脑缺血引起的一种局限性脑功能丧失，通常在24小时内完全缓解，不遗留重要神经功能缺陷。主要病因是脑动脉粥样硬化，亦可见于各种原因的动脉炎和心脏病。颈内动脉系统的脑缺血发作以病灶对侧的单瘫或偏瘫为常见，尤以上肢和面部为重，可伴有失语及精神症状。椎-基底动脉系统的脑缺血发作常见症状有眩晕、复视、构音障碍、吞咽困难、共济失调、单侧或双侧肢体瘫痪或感觉障碍等，至少两种以上症状共同出现。大脑后动脉供血不足可出现皮质盲，对侧同向偏盲。防治短暂性脑缺血发作，应针对每个人的病因，对发作次数多，考虑为微栓塞所致者，可慎重地选择抗凝治疗。主要病灶在颈部的动脉、颈内动脉颅内段或限于大脑中动脉主干者，可结合病人的具体情况考虑外科治疗。
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参考词条