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1)  atMDR
不典型多药耐药
2)  MDR
非典型多药耐药
1.
Reversal of BCRP Mediated-Atypical MDR Phenotype by Adenovirus-Delivered RNA Interference;
腺病毒加载的RNAi逆转BCRP介导的非典型多药耐药的研究
3)  multidrug resistance
多药耐药
1.
Function and mechanism of Baihua Sheshecao Injection reversing multidrug resistance of K562/ADM cells;
白花蛇舌草注射液逆转K562/ADM细胞多药耐药的作用和机制
2.
In Vitro study of the effect of cyclosporine A,tetrandrine and their combination on the reversion of multidrug resistance in leukemia cell line;
环孢菌素A、汉防己甲素及两者联合逆转K562/A02细胞的多药耐药
3.
Reversal of multidrug resistance of hepatocellular carcinoma by siRNA/mdr1;
RNA干扰逆转肝细胞癌多药耐药
4)  Multi-drug resistance
多药耐药
1.
The research of CIK cells reverse multi-drug resistance in vitro;
CIK细胞体外逆转多药耐药性的研究
2.
Relation between multi-drug resistance and apoptosis of HepG2/5-Fu;
人肝癌HepG2/5-Fu耐药细胞株多药耐药与凋亡关系的研究
3.
Expression of glutathione S-transferase π and multi-drug resistance associated protein in squamous cell carcinoma tissue;
皮肤鳞状细胞癌组织中谷胱甘肽-S-转移酶和多药耐药相关蛋白的表达
5)  Multidrug-resistance
多药耐药
1.
Expression of N-methylpurine-DNA-glycosylase in lung cancer H446/CDDP multidrug-resistance cell line;
N-甲基化嘌呤-DNA-糖基化酶在肺癌H446多药耐药细胞中的表达
2.
Study on the mechanisms of multidrug-resistance reversed by quercetin in HL-60/ADR;
槲皮素逆转HL-60/ADR多药耐药机制的研究
3.
Methods Myelodysplastic syndrome cell line(MUTZ-1) and a multidrug-resistance(MDR) erythroleukemia cell line(K562/A02) were cultured in medium of RPMI 1640 with 0.
L-1阿霉素的RPM I 1640分别培养MDS细胞株MUTZ-1和多药耐药(MDR)红白血病细胞株K562/A02。
6)  MDR
多药耐药
1.
The Mechanism on Reversion of MDR in Human Carcinoma Line with HCFU-SLNs;
卡莫氟固脂纳米粒对人大肠癌细胞多药耐药的逆转的机制研究
2.
Research on Inhibition of Cell Apoptosis and Multidrug Resistance (MDR);
细胞凋亡抑制与肿瘤多药耐药的研究进展
3.
Study on Effect Reversing MDR of Ligustrazine Liposomes on Human Erythroleukemia cell line K _(562) /ADM;
川芎嗪脂质体对人白血病细胞株K_(562)多药耐药逆转作用的研究
补充资料:安眠药、镇静药


安眠药、镇静药
HyPnoties,Sedatives

an而anyao、Zhenjingyao安眠药、镇静药Hypnotics,SedativeS蔡月刚上海医药工业研究院L概述··············……2.主要的安眠、镇静药2.1.醇类·········.··..……2.2.环状酞胺类·····……2.3.酸脉类···········……2.4.氨基甲酸醋类·”一2.5.苯二氮草类····……2.6.吩唯嗦类·······································……312.7.乙醇胺类·······································……312.8.二苯甲烷类······················,····……,··……312.9.叱咯酮类·······································……312.10.巴比妥类·······································……31参考文献················································……咒墓本参考文献··········································……33q八成J gJ广n占亡户nt了0‘2,﹄Q乙2,︺21.概述 安眠药又称催眠药,是一类对中枢神经系统产生抑制作用的药物,在应用适当剂量时,这种抑制作用能导致睡眠(见神经调节荆)。在较小剂量时可使紧张、焦虑和兴奋不安的患者安静下来,有思睡状态,但又不致入睡,这时就称为镇静药。大剂量的安眠药还可产生麻醉。实际上安眠药和镇静药并无明显的界限,而只有量的差别。有些类别的安眠药如使用特大剂量时还可引起昏迷和可能死亡。 失眠是一种不能得到良好睡眠的生理现象,表现为入睡困难、早醒、夜不成寐,即使暂时入睡,但醒后常感头晕脑胀、腰酸背痛,并无正常睡眠醒后的清新之感等等,总之,失眠患者常不能从睡眠中完全消除疲劳。失眠常与下列原因有关;如焦虑、心理障碍、精神上的打击、噪声(见噪声)和工作过度等等。长期失眠可造成中枢神经细胞功能失调,适当应用安眠药是必要的。 理想的安眠药应能使病人安然入睡,而在醒后不遗留任何不适感,并有正常工作的能力。一般患有失眠症的人,服用安眠药后即能获得类似生理性的睡眠,但多数醒后有精神萎靡不振等不良反应。如由于某些躯体疾病,如关节炎、神经痛和心绞痛等引起的失眠,则需首先进行病因性治疗口长期应用安眠药可产生依赖性和成瘾性。 目前对于睡眠发生的机理尚不清楚,但经过多年的努力,已经深入了一步。
说明:补充资料仅用于学习参考,请勿用于其它任何用途。
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