1) lung cancer cell
肺癌原代细胞
1.
Inhibition of lung cancer cells of primary culture and A549 cell line on the proliferation of tumor-infiltrating lymphocytes in vitro;
肺癌原代细胞和细胞系A549对肿瘤浸润性淋巴细胞体外增殖的抑制作用
2) pulmonary carcinoma cell/human pulmonary carcinoma clee LA/primary culture cell
肺癌细胞/人肺癌细胞LA、肺癌原代细胞
3) lung cancer primary cell
肺腺癌原代细胞
4) Lung cancer cell
肺癌细胞
1.
Polyamine analogue CPENSpm inhibits proliferation of human lung cancer cells by interfering polyamine metabolism;
多胺类似物CPENSpm通过干扰多胺代谢抑制肺癌细胞的增殖
2.
Analysis of C3orf1 gene over-expressed in human lung cancer cell line 95D with high metastatic potential;
高转移肺癌细胞株95D中特异表达的C3orf1基因分析
3.
China cobra venom active factor (CCVAF) inhibits VEGF and bFGF in lung cancer cell line H1299;
中华眼镜蛇毒组分抑制肺癌细胞H1299的血管生长因子VEGF和Bfgf
5) lung cancer cells
肺癌细胞
1.
Comparison of sodium selenite and all-trans retinoic acid on dose-effect relationship in lung cancer cells;
亚硒酸钠、维甲酸对肺癌细胞浓度效应关系的比较
2.
OBJECTIVE:To investigate the mechanism how the polarity of drugs affect the function of mitochondria of lung cancer cells.
目的:探讨药物极性对肺癌细胞线粒体功能的影响。
3.
The effects of serum concentration on growth inhibition of lung cancer cells by all trans retinoic acid(ATRA)were investigated.
研究不同浓度的血清对全反式视黄酸 (ATRA)抑制肺癌细胞生长的影响 。
6) Lung cancer
肺癌细胞
1.
According to the theory of traditional Chinese medicine, clinical practices andpast investigative results, we chose WeiMaiNing,berberine and HuangQi polysaccharide, whichhas the anti-lung cancer, anti-various tumors and immune regulation function, respectively, andconstituted the Chinese medicine comp.
本着中医治疗肿瘤“扶正与祛邪相结合”的基本原则,以“扶正培本、清热解毒”为治疗法则,根据临床实践经验和前人研究的结果,选用具有抗肺癌作用的威麦宁、抗多种肿瘤作用的小檗碱和具有免疫调节作用的黄芪多糖等三种成分组成中药复方,在整体、细胞、分子及基因水平上研究它的抗肺癌细胞增殖及抗肺癌转移作用,并深入探讨其作用的分子机制,为抗肿瘤转移药物的研制和开发及临床抗肿瘤转移治疗提供实验和理论依据。
2.
To explore effect of multidrug resistance mechanism, enzyme direct repair mechanism and nucleotide excision repair mechanism in lung cancer cells resistance to DDP.
目的:研究顺铂(DDP)对肺癌细胞耐药相关基因中的多药耐药相关蛋白1(MRP1)、O~6-甲基鸟嘌呤-DNA甲基转移酶(MGMT)、着色性干皮病A(XPA)mRNA表达水平的影响,探讨细胞膜蛋白介导的多药耐药机制,酶直接修复机制,核苷酸切除修复(NER)机制在肺癌细胞对DDP耐药过程中的作用。
3.
Methods: Lung cancer cell lines A549 and NCI-H446 were cultured in vitro.
结果: 1、MTT法显示OCT对肺癌细胞株A549的抑制作用随着浓度的增加,作用时间的延长,其抑制率增加(P﹤0。
补充资料：大细胞未分化型肺癌
大细胞未分化型肺癌
large cell undifferentiated carcinoma of lung
是支气管肺癌的一种类型。由大小不一的多边形细胞组成，呈实体巢状排列。癌细胞核大，核仁大而明显，胞浆丰富。可发生在肺门附近或肺边缘的支气管。治疗以手术、放疗或化疗综合治疗。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条