1) Bcl-2 gene family
Bcl-2基因家族
1.
The role of Bcl-2 gene family in testis germ cell apoptosis;
Bcl-2基因家族在睾丸生殖细胞凋亡中的作用
3) Bcl family gene
Bcl家族基因
4) Bcl-2 family
bcl-2家族
1.
Many related stimuli coming from intracellular or extracellular induce the activation of BH3(Bcl2-homology domains)-only protein,which regulates the formation of mitochondrial outmembrane permeabilization via members of Bcl-2 family,which results in the releasing of cytochrome c and Smac into the cytoplasm.
细胞内外各种刺激信号引起Bcl-2同源结构域3(Bcl-2 homology domain3,BH3)-only蛋白激活,通过Bcl-2家族(B celllymphoma 2)调控线粒体,使线粒体外膜渗透而导致细胞色素C及(second mitochondrial-derived activa-tor of caspase,Smac)流入胞质与凋亡活化因子-1(apoptosis activated factor-1,Apaf-1)及胱天蛋白酶-9(caspase-9)结合形成复合体。
2.
The expression of seven members of Bcl-2 family (including pro-apoptosis member of Bcl-2 family such as Bak, Bad, Bid, Bcl-xs and Bax, and anti-apoptosis ones such as Bcl-w and Bcl-2) and hepatitis B virus (HBV) proteins (such as, HBsAg, HBcAg and HbxAg) was detected in the tissues to texplore the significance of their expression.
应用免疫组织化学方法检测了34例肝癌组织及其相对应的癌旁组织,探讨了Bcl-2家族中七种基因(包括促凋亡基因Bak、Bad、Bid、Bax和Bcl-xs及抑凋亡基因Bcl-2、Bcl-w)和乙肝病毒三种抗原(包括HBsAg、HBcAg和HBxAg)在肝癌组织中的表达及意义,结果显示:在肝癌组织中HBsAg、HBcAg和HBxA的阳性率分别为58。
3.
Method Based on the immunohistochemical technology and electron microscope, We observed the relation between Bcl-2 family and the apoptosis in PC12 cells induced by dopamine.
目的研究Bcl-2家族的表达同多巴胺诱导的PC12细胞凋亡的相关性,探讨帕金森病(PD)神经元的死亡机制。
5) bcl-2 gene
bcl-2基因
1.
Effects of cadmium on the apoptosis of mouse spermatogenic cells and expression of bax and bcl-2 genes;
镉对小鼠生精细胞凋亡及bax和bcl-2基因表达的影响
2.
Inhibitory effects of shRNA expression vector on the expression of human ovarian carcinoma Bcl-2 gene;
RNAi表达载体对卵巢癌Bcl-2基因表达的抑制作用
3.
Effect of combination of marrow stromal cell and bcl-2 gene on neurological deficit and IGF-1 expression in rats with cerebral ischemia;
骨髓基质细胞与bcl-2基因对脑缺血大鼠疗效以及IGF-1表达影响的研究
补充资料：基因家族
分子式：
CAS号：
性质：真核生物的基因组中存在着的许多来源相同、结构相似、功能相关的基因。
CAS号：
性质：真核生物的基因组中存在着的许多来源相同、结构相似、功能相关的基因。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条