1) smoke inhalation injury
烟雾吸入性损伤
1.
To explore the role of apoptosis, apoptosis regulating genes in the pathogenesis and development of smoke inhalation injury.
探讨细胞凋亡及凋亡调控基因在大鼠烟雾吸入性损伤发生、发展中的作用及意义。
2) Smoke inhalation injury
烟雾吸入损伤
3) smoke inhalation injury
烟雾吸入伤
1.
Changes in the activities of antioxidases and content of glutathione of lung tissue in rat after smoke inhalation injury;
烟雾吸入伤大鼠肺组织抗氧化酶活性和谷胱甘肽含量变化
2.
The changes and its significances of interleukin-6 and interferon-α of lung tissue in rats after smoke inhalation injury;
烟雾吸入伤大鼠肺组织白介素-6和α-干扰素的变化及意义
3.
Objective To investigate the effect of massive bronchoalveolar lavage(MBL) with oxygenated perflurodecalin in early post injury stage on acute severe single lung smoke inhalation injury in dogs comparing with MBL with normal saline.
方法 建立犬左肺单侧急性重度烟雾吸入伤模型 ,伤后 1h对伤肺进行氧合氟碳液PFC或生理盐水大容量灌洗。
4) Inhalation injury
吸入性损伤
1.
Analysis of 20 cases of inhalation injury;
20例中重度吸入性损伤治疗分析
2.
Treatment of severe burn with inhalation injury;
特重烧伤伴吸入性损伤的治疗
3.
Management of extensive burn accompanying severe inhalation injury;
特大面积烧伤伴重度吸入性损伤的救治
5) smoke inhalation injury
吸入性损伤
1.
Objective To investigate the effects of inhaled 20 ppm NO on reactive oxygen species ( ROS) of polymorphonuclear (PMN) leucocyte in rat lung in early stage of smoke inhalation injury.
除了伤前值组大鼠外,其余各组均复制成重度烟雾吸入性损伤模型。
2.
Smoke inhalation injury was produced in all rat.
目的 研究表皮细胞生长因子 (EGF)对吸入性损伤所致大鼠肺组织水肿的治疗作用。
6) Smoke inhalation
烟雾吸入
1.
Objective: To establish an animal model of low saturation of blood oxygen with long-term and stabilization, used to assess smoke inhalation lung injury.
目的: 建立具有稳定、持久低血氧饱和度并且适用于研究烟雾吸入性肺损伤的动物模型。
2.
Methods The model of injury from smoke inhalation in SD rat was set up propotionally scaling-down according to real cabin and operator.
方法建立密闭舱内动物烟雾吸入模型,将36只SD大鼠完全随机分为对照组及烟雾吸入后1、6、24、72h和7d组,观察吸入损伤后不同时相点肺组织的病理改变,肺水含量及肺通透指数的变化,采用RT-PCR法检测大鼠肺组织SP-AmRNA的表达。
补充资料：吸入性损伤
吸入性损伤
inhalation injury
过去称为“呼吸道烧伤”，因此名词不够贴切，目前国内外统称为吸入性损伤。此因吸入火焰、干热空气、蒸气、有毒或刺激性烟雾或气体所致，因而有热力和化学性损伤。有些患者吸入有毒气体可致全身中毒。吸入性损伤多发生于密闭的房间或在火场中呼喊、奔跑。分三类：①轻度：烧伤在咽喉以上，口、鼻、咽黏膜充血水肿或苍白脱落，分泌增多，鼻毛烧焦，有刺激性咳嗽，吞咽困难或疼痛。②中度：损伤在大支气管以上，上述症状较重，有声嘶、呼吸困难、炭粒粉尘痰，可有干■音，气管切开后呼吸困难可改善。③重度：损及小支气管，上述症状严重，呼吸困难不因气管切开而改善，肺水肿出现较早，肺呼吸音弱，有干湿■音。处理原则：①尽早液体复苏，需要补足血容量；②保持呼吸道通畅和湿润，包括气管插管、气管切开，超声雾化吸入，解痉药物，气道内点滴生理盐水等；③必要时用呼吸机；④防治感染。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条