1) mitochondrial ATP-sensitive potassium channel
线粒体膜ATP敏感钾通道
1.
[Objective] To evaluate the early and the delayed protection of diazoxide ( a mitochondrial ATP-sensitive potassium channel opener ) on myocardial ischemia- reperfusion injury the effects of ischemia-reperfusion on hemodynamics , electrophysiologic basis,myocardial ultrastructure, myocardial infarct size and cardiomyocytic apoptosis were observed in ameasthetized rabbit myocardium.
【目的】在麻醉家兔心肌缺血-再灌注(IR)模型上，观察IR和线粒体膜ATP敏感钾通道(mitK_(ATP))开放剂二氮嗪预处理早期及延迟相对血流动力学、心电生理、心肌梗塞范围、心肌酶学、心肌超微结构及心肌细胞凋亡的影响。
2) mitochondrial ATP-sensitive potassium channel
线粒体ATP敏感性钾通道
1.
Cardioprotective effects of Mitochondrial ATP-sensitive potassium channel opener diazoxide on ischemic myocardial cells;
线粒体ATP敏感性钾通道开放剂二氮嗪对大鼠缺血心肌细胞的保护作用
2.
Objective To investigate the protection of preconditioning by mitochondrial ATP-sensitive potassium channel opener from simulated ischemia/reperfusion injury in cultured cardiac myocytes.
目的探讨线粒体ATP敏感性钾通道开放剂二氮嗪预处理对大鼠心肌细胞线粒体功能保护作用。
3.
AIM: To demonstrate the effect of mitochondrial ATP-sensitive potassium channel opener,Diazoxide,on glutamate uptake activity of astrocyte.
目的:研究线粒体ATP敏感性钾通道(mito-chondrial ATP-sensitive potassium channel,mitoKATP)开放剂二氮嗪(diazoxide)对星形胶质细胞摄取谷氨酸(glutamate)的影响。
4) mitochondrial ATP sensitive potassium channel
线粒体ATP敏感性钾通道
1.
More and more studies are shown that mitochondrial ATP sensitive potassium channel plays an import role,acting as an end-effector,in these pathways.
缺血预处理对心肌的保护机制十分复杂,一般公认为线粒体ATP敏感性钾通道(mitoKATP)是心肌缺血预处理保护机制的终末效应器之一。
5) Mitochondrial ATP-sensitive potassium channels
线粒体ATP敏感性钾通道
1.
Mitochondrial ATP-sensitive potassium channels are located in the inner mitochondrial membrane (mio KATP), which are regulated by the ATP concentration in cytosole.
线粒体ATP敏感性钾通道是一类存在于线粒体内膜并受胞内ATP浓度调节的钾离子通道。
2.
Objective To determine the relationship between the protection of bilobalide against focal cerebral ischemia-reperfusion injury and mitochondrial ATP-sensitive potassium channels(mito K-ATP) in vivo.
目的研究银杏内酯抗脑缺血-再灌注损伤作用与线粒体ATP敏感性钾通道(mito K-ATP)开放的关系。
6) mitoKATP
线粒体ATP敏感性钾通道
1.
AIM: To study the influence of glipizide on myocardial mitochondrial ATP-sensitive potassium channels (mitoKATP) in normal and diabetic rats by measuring the changes of mitochondrial volume.
目的:探讨格列吡嗪(Glip)对正常及糖尿病大鼠心肌线粒体ATP敏感性钾通道(mito KATP)的影响。
2.
Objective To explore the effect of bcl-2 in attenuation ischemia-reperfusion injury(I/RI) in liver graft in mice induced by mitochondrial ATP-sensitive potassium channel(mitoKATP) opener diazoxide and the feasibility of reducing ischemia reperfusion injury of graft liver by diazoxide.
目的研究bcl-2在线粒体ATP敏感性钾通道(mitoKATP)开放剂二氮嗪(DZ)诱导的抗小鼠移植肝脏缺血再灌注损伤中的作用,探讨应用二氮嗪减轻移植肝缺血再灌注损伤的可行性。
补充资料：线粒体膜
分子式：
CAS号：
性质：系双层膜结构，此两层膜的结构和组成都不相同。外层膜类脂含量高，有明显的酶互补体。两层膜的渗透性能也不同，外层膜能允许低分子量化合物和多种蛋白质自由地渗透而内层膜对许多离子、低分子量化合物和蛋白质具有渗透障碍。内层膜具有特殊的穿膜运输系统。
CAS号：
性质：系双层膜结构，此两层膜的结构和组成都不相同。外层膜类脂含量高，有明显的酶互补体。两层膜的渗透性能也不同，外层膜能允许低分子量化合物和多种蛋白质自由地渗透而内层膜对许多离子、低分子量化合物和蛋白质具有渗透障碍。内层膜具有特殊的穿膜运输系统。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条