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1)  Cardiac mast cells
心脏肥大细胞
2)  Cardiac hypertrophy
心脏肥大
1.
There has recently been a notable increase in the amount of research into the signal transduction pathway among pathogens of cardiac hypertrophy.
心脏肥大是心肌细胞对高血压、瓣膜病、急性心肌梗死及先天性心脏病等常见临床疾病的一种基本应答,是影响心血管疾病病死率和发病率的独立危险因素,其发病机制中信号转导通路的研究是当前研究热点。
2.
Hemodynamic overload, namely mechanical stress, is a major cause of cardiac hypertrophy.
血流动力超荷即机械应力是引起心脏肥大的主要原因,它能通过许多不同的信号途径诱导心脏肥大的发生,弄清楚机械应力诱导心脏肥大的机理,将有助于阐明充血性心力衰竭的机理。
3)  myocardial hypertrophy
心脏肥大
1.
Twenty rats with exercise and pressure_overload induced myocardial hypertrophy were taken as objects,the parameters of myocardial cells,specific surface and specific membrane of mitochondria were measured to ulteriorly find out the morphological differences between endurance training induced and pathological myocardial hypertrophy.
为了解耐力运动性与病理性心脏肥大在形态学上的差异 ,本文在大鼠游泳训练心肌肥大和肾血管性高血压肥大模型上 ,测量了各组心肌细胞参数及心肌线粒体比表面与内膜比膜面。
4)  Heart hypertrophy
心脏肥大
5)  cardiomyocyte hypertrophy
心肌细胞肥大
1.
Effects of exogenous Apelin on cardiomyocyte hypertrophy;
Apelin对心肌细胞肥大的影响
2.
Study of cross-talk between stat family and smads pathway mediated cardiomyocyte hypertrophy in cultured neonatal rat in vitro
新生大鼠心肌细胞通过STAT和Smads信号交叉对话介导心肌细胞肥大的体外研究
3.
OBJECTIVE To observe effects of Sodium tanshinone IIA sulfonate(STS) on angiotensin II(Ang Ⅱ)-induced cardiomyocyte hypertrophy and expressions of phosphorylated p38(p-p38) and mitogen-activated protein kinase phosphatase-1(MKP-1).
目的观察丹参酮ⅡA磺酸钠(Sodium tanshinoneⅡA sulfonate,STS)对血管紧张素Ⅱ(angiotensinⅡ,AngⅡ)诱导的心肌细胞肥大反应及p-p38,MKP-1表达的影响。
6)  Cardiac hypertrophy
心肌细胞肥大
1.
The Effect of Erythropoietin on cardiac hypertrophy
促红细胞生成素促心肌细胞肥大的实验研究
2.
Objective: to study the inhibition effect of simvastatin on cardiac hypertrophy induced by myocardial infarction,and to investigate whether simvastatin prevent cardiac hypertrophy through attenuation of RhoA/ROCK and cyclin D1.
目的:观察辛伐他汀对大鼠心肌梗死后心肌细胞肥大的抑制作用,探讨小G蛋白RhoA激酶(ROCK)/细胞周期蛋白D1(Cyclin D1)途径在辛伐他汀抑制心肌细胞肥大中的信号转导作用。
3.
However,little is known about the role of Epo in the development of cardiac hypertrophy.
有关Epo对心肌细胞肥大影响的报导在国内外非常有限。
补充资料:心脏肥大


心脏肥大


多指心肌肥厚引起的心脏增大。参见"心脏扩大"。
说明:补充资料仅用于学习参考,请勿用于其它任何用途。
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