Methods The acute hemorrhagic and necrotic pancreatitis (AHNP) model was induced by the standard retrograde infusion of bilio-pancreatic duct with 4% sterile sodium taurocholate solution in Sprague-Dawley rats.

Conclusion Acute hemorrhagic enteritis has complex clinical symptoms,high misdiagnosis rate of early complications and mortalit.

Effects of tetrandrine on inflammatory cytokines in rats with acute hemorrhagic necrotizing pancreatitis;

Effect of tetrandrine on production of nitrogen monoxide of acute hemorrhagic necrotizing pancreatitis in rat;

Experimental research of L-arginine in the treatment of acute hemorrhagic necrotizing pancreatitis in early stage;

Protective effects of cyclosporin A on brain damage of acute necrotizing pancreatitis in rats;

Objective To evaluate whether liver plays a protecting role in rats with acute necrotizing pancreatitis(AP),and the role of the liver in mediating serum IL-6 level.

Changes in Toll-like receptor 2 and 4 gene expression in hepatic injuries complicated in acute hemorrhage necrotizing pancreatitis rats;

Pathomorphological observation of liver injury caused by experimental acute hemorrhagic and necrotic pancreatitis and apoptotic cell death of hepatocytes in liver;

To investigate the role of endogenetic NO in the pathogenesis of acute hemorrhagic and necrotic pancreatitis, AHNP was induced in rats (300～350 g) by intraductal administration of 5% sodium taurocholate.

Objective: To observe the pathomorphological changes of pancreatic microcirculation in the early phase of acute hemorrhagic and necrotic pancreatitis.