1) matrix secreted
系膜基质增生
1.
Objective To observe the effect of the prescription on the progress of mesangial cell proliferation and matrix secreted induced by AGEs.
结果1×10-3、1×10-2、1×10-1mg/mL的中药复方提取液均能抑制AGEs培养下的鼠肾小球系膜细胞的增殖;能减少AGEs作用下的细胞上清液中羟脯氨酸含量,抑制系膜基质增生。
2) mesenterium proliferative
系膜增生
1.
Results Children suffered from different clinical types of single hematuria mainly presented mesenterium proliferative changes,few children appeared focal /segmental cell proliferation,necrosis,fibrosis and glomerular sclerosis(5 cases,15.
结果不同临床类型的单纯性血尿患儿肾组织病理主要表现为系膜增生性改变;少数伴局灶/节段性细胞增生、坏死、纤维化和肾小球硬化(5例,占15。
2.
ResultsChildren suffered from different clinical types of single hematuria mainly presented mesenterium proliferative changes,and the percentage of minor and diffuse proliferation were 37.
结果不同临床类型的单纯性血尿患儿肾组织病理主要表现为系膜增生性改变,其中轻度增生和弥散性增生分别占37。
3) mesangial matrix
系膜基质
1.
Effect of captopril on mesangial matrix of mesangial proliferative glomerulonephritis in rabbits;
卡托普利对系膜增殖型肾炎家兔系膜基质的影响
2.
Effects of benazepril on the accumulation of mesangial matrix and the excretion of proteinuria in mesangial proliferative glomerulonephritic rats;
贝那普利对抗Thy1.1大鼠系膜基质积聚及尿蛋白的影响
4) mesangial proliferative
系膜增生性
1.
Methods: The Thy1 model of experimental mesangial proliferative glomerulonephritis was induced by intravenous injection of rabbit anti-thymocyte plasma.
目的 :探讨周期素激酶抑制剂 P2 7在系膜增生性肾炎模型 Thy1肾炎大鼠系膜细胞 (MC)增生中的作用。
6) MsPGN
系膜增生性肾炎
1.
Comparative study of interference of No1 therapy of chronic nephritis on microstructure of immune molecule in mice with MsPGN;
慢肾1号方对小鼠系膜增生性肾炎免疫分子等超微结构干预的对照性研究
2.
Effect of MACC5 isolated from traditional Chinese medicine on the rats with MsPGN induced by anti-Thy1.1;
中草药提取物MACC5对大鼠抗Thy1.1系膜增生性肾炎模型的疗效观察
3.
Objective To study the changes of AQP-2 and ADH of rats with damp-heat type of MsPGN and the mechanism of retention of damp-heat in the interior.
目的研究系膜增生性肾炎(MsPGN)湿热型大鼠水通道蛋白-2(AQP-2)的表达及抗利尿激素(ADH)的变化及湿热蕴郁的机制,并探讨慢肾蛋白停对其的影响及作用机理。
补充资料：卵巢间质增生和卵泡膜增生
卵巢间质增生和卵泡膜增生
单纯卵巢间质增生所致双侧卵巢增大，即‘卵巢间质增生’；有黄素化间质细胞者称‘卵泡膜增生’。多发生于绝经后，可能因绝经后下丘脑-垂体功能紊乱，卵巢间质对垂体促性腺激素的一种增生性反应。常伴发糖尿病、高血压、肥胖、和甲状腺功能减退。临床上出现雌激素或雄激素过高现象。可伴子宫内膜增生甚至内膜癌等病变。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条