1) Galactosamine
[英][ɡə,læk'təusə,mi:n] [美][,ɡælək'tosə,min, ɡəlæk-]
半乳糖胺
1.
Influence of N,N -diacetyl-L-cystine on D-galactosamine/ lipopolysaccharide induced immunological liver failure in mice;
N,N’-二乙酰-L-胱氨酸对半乳糖胺联用脂多糖引起小鼠免疫性肝衰竭的作用(英文)
2) D galactosamine
半乳糖胺
1.
Methods In the process of establishing rat liver fibrosis induced by D galactosamine (D Gal) and dimethylnitrosamine (DMN), we observed the effects of oxymatrine on the following parameters: liver/weight index, serum and tissue biochemical indexes, TGFβ1 mRNA liver hydroxyproline expression content, electron microscopic changes and tissue pathology.
方法 建立半乳糖胺和二甲基亚硝胺大鼠肝纤维化模型 ,观察氧化苦参碱 (3 0mg/kg、90mg/kg)预防前后肝指数、血及肝组织生化、羟脯氨酸含量、TGFβ1mRNA表达、电镜及病理组织学改变。
2.
Methods By establishing D galactosamine induced rat liver fibrosis, we observe the effect of oxymatrine on liver/weight index , serum and tissue biochemical indexes, content of liver hydroxyproline , expression of TGFβ1 mRNA,changes of electron microscopy and tissue pathology .
方法 采用半乳糖胺诱导的大鼠肝纤维化模型 ,观察氧化苦参碱 ( 30mg/kg、90mg/kg)干预前后肝指数、血及肝组织生化、羟脯氨酸含量、TGFβ1mRNA表达水平、电镜及病理组织学改变。
3) D-galactosamine
半乳糖胺
1.
Prophylactic and therapeutic effect of oxymatrine on D-galactosamine-induced rat liver fibrosis;
氧化苦参碱防治半乳糖胺诱导大鼠肝纤维化的实验研究
2.
The experimental hepatic lesion of mouse were induced by intraperitoneal injection of D-galactosamine (500mg·kg￣(-1)).
以Ｄ－半乳糖胺（５００ｍｇ·ｋｇ~（－１））给予小鼠腹腔注射２４～４８ｈ后，导致实验性肝病变，观察可见肝细胞组织酶反应减弱，超微结构严重受损。
4) D-GalN
D-半乳糖胺
1.
Objective To study the effect of Yugan Tablet on acute liver injuries induced by D-galactosamine(D-GalN)in mice.
目的研究愈肝片对D-半乳糖胺(D-GlaN)所致小鼠急性肝损伤的影响。
2.
Methods: Two acute hepatic injury models induced by carbon tetrachloride (CCl4) and D-galactosamine (D-GalN) in mice were used.
方法:CCl_4和D-半乳糖胺小鼠肝脏损伤模型,以测定血清谷丙氨酸转换酶(ALT)、谷草氨基酸转换酶(AST)和肝匀浆丙二醛(MDA)含量为指标观察丹参酮ⅡA对肝脏损伤的保护作用。
5) D-galactosamine
D-半乳糖胺
1.
Pathogenesis of D-galactosamine(D-GalN)-and Lipopolysaccharides(LPS)-induced Fulminant Hepatic Failure
D-半乳糖胺和脂多糖介导大鼠急性肝衰竭的机理研究
2.
MethodsChemical liver injury models were made by D-galactosamine and Carbon tetrachloride and ALT,AST activities and MDA were detemined.
方法分别采用CCl4和D-半乳糖胺作为化学毒物制成急性肝损伤模型,采用赖氏法测定各组动物血清丙氨酸氨基转移酶(ALT)和血清天门冬氨酸氨基转移酶(AST)的活性,并测定肝匀浆MDA含量。
3.
METHODS: Acute hepatic injury was induced by intraperitoneal(ip) injection of carbon tetrachloride and D-galactosamine,respectively.
方法:分别采用四氯化碳(CC l4)和D-半乳糖胺(D-galactosam ine,D-Gal)腹腔注射造成大鼠急性肝损伤,测定血清生化指标,并测体重和肝脏重、脾重和胸腺重,求脏器系数。
6) D-aminogalactose
D半乳糖胺
补充资料：D-半乳糖胺
分子式：
CAS号：
性质：又称2-氨基半乳糖，软骨糖胺。一种半乳糖的氨基衍生物，存在于糖脂和硫酸软骨质(chondroitin sulfate)中。可以来苏糖、氨为原料制取。其盐酸盐结晶，熔点180℃(分解)。旋光度+93°(1%，水)，用于生化研究。
CAS号：
性质：又称2-氨基半乳糖，软骨糖胺。一种半乳糖的氨基衍生物，存在于糖脂和硫酸软骨质(chondroitin sulfate)中。可以来苏糖、氨为原料制取。其盐酸盐结晶，熔点180℃(分解)。旋光度+93°(1%，水)，用于生化研究。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条