1) oxygen-glucose deprivation
缺糖、缺氧
1.
Aim:To observe the protective effects of protein transduction domain-kallikrein(PTD-kallikrein)on neurons incubated in condition of oxygen-glucose deprivation(OGD)/recovery,and to explore the protective mechanism.
目的:观察蛋白质转导域-激肽释放酶(PTD-kallikrein)对缺糖、缺氧(OGD)再复糖、复氧培养的神经元保护作用,探讨其可能的作用机制。
2) Oxygen-glucose deprivation
缺糖缺氧
1.
The protective effect of LY367385 on impairment of cultured mouse cerebral cortical neurons induced by sodium glutamate or oxygen-glucose deprivation;
LY367385对谷氨酸钠及缺糖缺氧引起的培养小鼠大脑皮层神经元损伤的保护作用
3) Oxygen-glucose deprivation (OGD)
缺糖缺氧（OGD）
4) oxygen-glucose deprivation
缺糖缺氧损伤
5) protective effect against OGD-induced injury
抗缺糖缺氧损伤
6) Oxygen glucose deprivation
缺糖/缺氧性损伤
补充资料：缺氧缺血性脑病
缺氧缺血性脑病
hypoxic ischemic encephalopathy,HIE
发生在围产儿，尤易发生在窒息足月儿的疾病。脑组织以水肿、软化、坏死和出血为主要病变。重者有后遗症。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。