1) MUTZ-1 cell line
MUTZ-1细胞株
2) MuTz-1 cell
MuTz-1细胞
1.
Methods The growth inhibition of MuTz-1 cell induced by CDA-2 was detected by MTT;the expressions of P15INK4B,DNA methyltransferases(DNMT)1,DNMT3A and DNMT3B gene on mRNA level were detected by RT-PCR;the methylation state of p15INK4B gene in MuTz-1 cells was detected by methylation specific PCR(MSP).
结果CDA-2呈剂量依赖性抑制MuTz-1细胞生长;伴随CDA-2剂量增加,p15INK4B基因甲基化程度逐渐减弱,其mRNA表达逐渐增加;且CDA-2可显著降低MuTz-1细胞中DNMT1和DNMT3BmRNA的表达。
2.
To investigate the mechanisms of the telomerase regulations during the apoptosis of the human MDS-RAEB cell line MUTZ-1 cells induced by arsenic trioxide (As_2O_3), telomerase activity was detected by TRAP-ELISA and the expressions of mRNAs of hTERT, TRF1(TTAGGG repeat binding factor 1), TRF2(TTAGGG repeat binding factor 2), bcl-2, and bax genes were detected by RT-PCR.
为了研究三氧化二砷(As2O3)诱导人类骨髓增生异常综合征难治性贫血伴原始细胞过多型(MDS-RAEB)细胞株MUTZ-1细胞凋亡的端粒酶调控机制,采用端粒重复序列扩增-酶联免疫吸附试验(TRAP-ELISA)检测端粒酶活性;RT-PCR法检测端粒酶逆转录酶(hTERT)、TRF1(TTAGGGrepeatbindingfactor1)、TRF2(TTAGGGrepeatbindingfactor2)、bcl-2、bax等基因mRNA水平的表达;磷脂酰丝氨酸(PS)转位等方法检测细胞凋亡。
3) MUTZ-1 cells
MUTZ-1细胞
1.
Inhibition effect of arsenic trioxide on the growth of human MDS cell line MUTZ-1 cells;
三氧化二砷对人骨髓增生异常综合征细胞株MUTZ-1细胞的作用
2.
Methods: The apoptosis of MUTZ-1 cells induced by fludarabine was studied by transmission electron microscope,MTT assay,DNA ladder test,flow cytometry and RT-PCR method.
目的:在体外研究磷酸氟达拉滨(fludarab ine)对人骨髓增生异常综合征细胞株MUTZ-1细胞的作用以及可能的作用机制。
4) myelodysplastis syndrome MUTZ?1 cell line
骨髓异常增生综合征细胞株MUTZ-1
6) THP-1 cell line
THP-1细胞株
补充资料：细胞株
分子式：
CAS号：
性质：用单细胞分离培养法或克隆形成法从原代培养或从细胞系所选出的细胞群，称细胞株。一个细胞株应具有特定的生物学性质和标记并持续存在。
CAS号：
性质：用单细胞分离培养法或克隆形成法从原代培养或从细胞系所选出的细胞群，称细胞株。一个细胞株应具有特定的生物学性质和标记并持续存在。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条