1) Muscle atrophy
肌肉萎缩
1.
Purpose To study limb immobilization influencing on denervated muscle atrophy.
结论 肢体制动加速了失神经支配肌肉萎缩的发展。
2.
This paper reviews two main signaling pathways in order to introduce the key role of Akt in it,one is Akt1/FOXOs/MAFbx/MuRF1 signaling path- way which associated with muscle atrophy;the other is PI3K/Akt/mTOR/S6K1 signaling pathway which associated with muscle hypertrophy.
综述与骨骼肌质量变化相关的两个主要信号通路:一方面是与肌肉萎缩相关的Akt1/FOXOs/MAFbx/MuRF1信号通路;另一方面为与肌肉肥大相关的PI3K/Akt/mTOR/S6K1信号通路,从两方面介绍Akt在骨骼肌质量变化中所处的核心地位。
3.
Methods: In order to establish the cell model of using GCs to cause the muscle atrophy,we examined the total protein of the C2C12 muscle tube which was fed with Dexamethasone(DEX,100 nMs) for 1 to 4 days using BCA method.
目的:建立高剂量或长期服用糖皮质激素会引起肌肉萎缩的细胞模型。
2) muscular atrophy
肌肉萎缩
1.
Methods: Eighteen rats were randomly divided into 3 groups:control group,muscular atrophy group and intervention group.
方法:采用悬吊方法复制肌肉萎缩模型,SD雄性大鼠18只随机分成对照组、模型组和电刺激组,每组6只。
4) withering syndrome
肌肉萎缩症
1.
Test on drugs prevention and cure of withering syndrome of Haliotis Diversicolor Reeve
杂色鲍“肌肉萎缩症”的药物防治试验
5) disuse amyotrophy
肌肉废用性萎缩
1.
Prevention of disuse amyotrophy of lower extremities by exercises of isometric muscles contraction after inmobilized with plaster spica of hip;
髋人字石膏制动下肢等长收缩训练预防肌肉废用性萎缩
6) muscle disuse atrophy
废用性肌肉萎缩
1.
vastus lateralis after the subjects were immobilized on their right legs for two weeks to disuse atrophy then supplied creatine and training for ten weeks and to measure Myogenic Regulation Factors(MRFs)MyoD?Myogenin and Myf-5 ,to discuss the molecular mechanism which creatine could accelerate the rehabilitation of muscle disuse atrophy.
研究结果显示 :补充肌酸使股外侧肌废用性肌肉萎缩后恢复期各亚型肌纤维增粗 ,Ⅱ型肌纤维较Ⅰ型肌纤维增粗显著。
补充资料：Berardinelli脂肪萎缩综合征
Berardinelli脂肪萎缩综合征
为常染色体隐性遗传。表现：生长及成熟增快，手足增大，阴茎增大，但雄激素及促性腺激素均未增加，垂体及其所及的内分泌腺均接近正常；而下丘脑-释放因子相对升高。肌肉肥大含糖原过多，早年即出现脂肪缺乏。皮肤粗糙，色素沉着，尤其腋下更显著，有不同程度的黑色棘皮症。多毛，头发弯曲。浅表静脉扩张。肝肿大，有过量脂肪及糖原沉积，最后可致肝硬变。高脂血症，胰岛素增高，可能发生抗胰岛素性无酮体性高糖血症。糖尿病、肝硬化及食管静脉曲张可成为致死的并发症。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条