1)  Hypeluricemia
高尿酸
1.
Investigation of Clinical Correlative Factors of Hypeluricemia and Coronary Disease in Middle and Old-aged;
中老年人高尿酸血症与冠心病的临床相关因素探讨
2)  hyperuricemia
高尿酸血症
1.
A Study of Relationship between Hyperuricemia and Risk Factors of Coronary Disease;
高尿酸血症与冠心病危险因素关系的探讨
2.
Analysis of correlationship between hyperuricemia and metabolic syndrome;
高尿酸血症与代谢综合征的相关性分析
3.
Relationship between hyperuricemia and serum adiponectin,serum leptin and adiponectin-leptin ratio in elderly males;
老年男性高尿酸血症与血清脂联素、瘦素水平及其比值的相关性研究
3)  hyperuricaemia
高尿酸血症
1.
Clinical trial of cortex fraxini total coumarin in treating primary hyperuricaemia;
秦皮总香豆素治疗原发性高尿酸血症临床研究
2.
Efficacy and safety of domestic benzbromarone for the treatment of type 2 diabetes and hyperuricaemia;
国产苯溴马隆治疗2型糖尿病合并高尿酸血症的疗效与安全性
3.
Efficacy of domestic benzbromazone in treatment of 50 patients with hyperuricaemia;
国产苯溴马隆治疗高尿酸血症50例
4)  hyperuricacidemia
高尿酸血症
1.
Symptomatology study of Chinese medicine in quail with hyperuricacidemia;
鹌鹑高尿酸血症中医证候学研究
2.
Pathogenesis of hyperuricacidemia in quail model;
鹌鹑高尿酸血症模型发病机理研究
5)  Primary hyperuricemia
高尿酸血症
1.
Discussion on syndrome identifying and treatment of primary hyperuricemia;
原发性高尿酸血症辨治初探
2.
In order to discover a new agent to treat Primary hyperuricemia and to observe the effect of lipantyltreating hyperuricemia,we devide 89 cases with primary hyperuricemia into 2 groups.
为了探索治疗高尿酸血症的新药，观察力平脂的降血尿酸作用，将确诊原发性高尿酸血症患者８９例，随机分成二组，一组用力平脂治疗（称治疗组），另一组用别嘌呤醇治疗（称对照组）。
3.
Methods The patients and their first-degree relationsfrom 20 primary hyperuricemia and gout pedigrees underwent an oral 75gglucose test(OGTT) and were determined serum uric acid(SUA),60 cases ashyperuricemia and gout group (HG group) who accorded with diagnosis standardwere selected;60 subjects of similar age and sex with HG group in the controlgroup were selected from this locality.
目的探讨家系原发性高尿酸血症和痛风患者血尿酸对胰岛β细胞功能的影响。
6)  hyperuricaemia,hyperuricemia
高尿酸血<症>
参考词条
补充资料：高尿酸血症


高尿酸血症
hyperuricemia

人体核蛋白中嘌呤化合物的终末代谢产物——尿酸在血中浓度增多男性＞416μmol/L(7mg/dl），女性＞357μmol/L(6mg/dl）。其发病机制包括：①尿酸生成过多：此或为嘌呤核苷酸代谢途中某些酶缺陷所引起；或为体内尿酸生成原料过多(如骨髓增殖性疾病或肿瘤造成体内大量细胞增殖和破坏时）所造成。②尿酸排泄障碍：2/3～3/4尿酸由肾脏排泄，为此肾排泄尿酸障碍(如肾功能不全）也可导致高尿酸血症。上述酶缺陷造成的高尿酸血症称为原发性高尿酸血症，其他均称为继发性高尿酸血症。高尿酸血症可引起痛风性关节炎、痛风石及高尿酸血症肾病(包括急性或慢性高尿酸肾病及尿路尿酸结石）。治疗应抑制尿酸合成(如服用别嘌呤醇），促进尿酸排泄(如服用丙磺舒，明显高尿酸尿症及肾小管梗阻者慎用），并碱化尿液(如服小苏打，控制尿pH值6～6.5范围）。尚应控制含高核蛋白的饮食及多饮水。
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