1) 3T3-L1 adipocytes
3T3-L1脂肪细胞
1.
Molecular mechanism of insulin resistance induced by free fatty acids in 3T3-L1 adipocytes through targeting nuclear-kB p65;
游离脂肪酸刺激核因子NF-kBp65核转位诱导3T3-L1脂肪细胞胰岛素抵抗的分子机制
2.
Expression of adiponutrin mRNA in mature 3T3-L1 adipocytes;
Adiponutrin mRNA在分化成熟的3T3-L1脂肪细胞中的表达
3.
Continuously activated Akt inhibits adiponectin secretion in 3T3-L1 adipocytes;
持续激活的Akt减少了3T3-L1脂肪细胞脂联素蛋白的表达
2) 3T3-L1 adipocyte
3T3-L1脂肪细胞
1.
Establishment of GOD-POD assay in a minimal way and application to glucose metabolism of 3T3-L1 adipocyte and HepG2 cell in vitro;
GOD-POD法微量化测定方法的建立及其在3T3-L1脂肪细胞和HepG2细胞糖摄取中的应用
2.
Akt regulating adiponectin secretion in 3T3-L1 adipocytes;
蛋白激酶B对3T3-L1脂肪细胞脂联素蛋白质表达的调控
3.
This paper establishes the insulin resistance model of 3T3-L1 adipocytes.
建立3T3-L1脂肪细胞IR模型,葡萄糖氧化酶法测定葛根素处理48 h后培养液中葡萄糖残存量、铜试剂显色法测定游离脂肪酸浓度。
3) 3T3-L1 preadipocyte
3T3-L1前脂肪细胞
1.
Obestatin inhibits proliferation and differentiation of 3T3-L1 preadipocytes;
Obestatin抑制3T3-L1前脂肪细胞的增殖与分化
2.
Angiopoietin-like protein 4(ANGPTL4) gene expression during the period of 3T3-L1 preadipocyte differentiation
ANGPTL4基因在3T3-L1前脂肪细胞诱导分化过程中表达水平的变化
3.
The differentiation of 3T3-L1 preadipocyte,HL-60,K562 and B16 cells were assessed by microscopy and Oil red O staining,NBT reduction and phagocytosis,haemoglobin(Hb) content and melanin content,respectively.
方法:SRB法和乳酸脱氢酶释放法检测B16细胞活性;油红染色法检测3T3-L1前脂肪细胞的分化;NBT还原能力和细胞吞噬功能法检测HL-60细胞的分化;血红蛋白含量法检测K562细胞的分化;黑色素含量法检测B16细胞的分化。
4) 3T3-L1 Preadipocytes
3T3-L1前脂肪细胞
1.
Effect of emodin on differentiation induction of 3T3-L1 preadipocytes
大黄素对3T3-L1前脂肪细胞诱导分化的作用
2.
【Objective】 The aim of this study is to investigate the effect of different concentrations of sporamin on differentiation and proliferation of 3T3-L1 preadipocytes,providing a theoretical basis for the development of prevention and treatment of obesity and anti-diabetes reagent in food industry.
【目的】探讨甘薯sporamin蛋白对3T3-L1前脂肪细胞分化与增殖的影响,为开发预防和治疗肥胖、糖尿病的保健食品提供理论依据。
3.
Objective To investigate the effect and underling mechanism of genistein(GEN)on adipogenesis in 3T3-L1 preadipocytes.
目的观察染料木黄酮(genistein,GEN)对3T3-L1前脂肪细胞分化的影响,探讨GEN抑制3T3-L1细胞分化的作用及分子机制。
6) 3T3–L1 adipocyte
3T3–L1脂肪细胞
1.
Methods After utilizing dexamethasone and insulin to establish insulin-resistant modle by induced 3T3–L1 adipocyte, the levels of tumor necrosis-alpha(TNF–α),interleukin–6(IL–6) and C–reactive protein (CRP) of cell supernatant were detected.
目的:探讨糖耐康颗粒对胰岛素抵抗3T3–L1脂肪细胞炎症因子的影响。
补充资料：羊水脂肪细胞含量
羊水脂肪细胞含量
主要借助羊水中脂肪细胞比例判定胎儿皮肤成熟的情况。随着妊娠进展，胎儿皮脂腺逐渐成熟，羊水中含脂肪细胞出现率逐渐增多。妊娠34周前，羊水中脂肪细胞少于1%，于孕34～37周，所占比例为1%～10%，孕38～40周为20%左右。如果羊水中脂肪细胞比例在20%以上，可认为胎儿已成熟，由于该方法估计胎儿成熟度的准确性较低，目前很少应用。羊水中脂肪细胞含量测定方法为：取羊水沉渣均匀涂于玻璃片上，采用硫酸尼罗蓝染色，可发现有两种类型细胞：一种胞浆呈均匀蓝色，为胎儿上皮细胞，另一种呈橘黄染色，即来自胎儿皮脂腺的脂肪细胞。镜下数200个细胞，计算含脂肪细胞的百分数。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条