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1)  vascular hyporeactivity
血管低反应性
1.
Relationship between calcium sensitivity regulation and vascular hyporeactivity following hemorrhagic shock;
血管平滑肌细胞钙敏感性调控与休克血管低反应性发生的关系
2.
Effect of melatonin on vascular hyporeactivity of rats in septic shock;
5-甲氧色胺对感染性休克大鼠血管低反应性的作用
3.
The occurrence of vascular hyporeactivity after severe trauma or shock has been shown to have important roles in the incidence, development, and the outcome of shock and interfered with the therapy of shock, but its mechanisms remain unclear.
严重创伤/休克后出现的血管低反应性对休克的发生、发展和转归以及休克治疗有着重要影响,其确切机制尚未完全阐明。
2)  vascular hyporesponsiveness
血管低反应性
1.
Aim To study the effects of genistein on vascular hyporesponsiveness of superior mesenteric artery (SMA) in hemorrhagic shock rat and its mechanism.
结果失血性休克导致大鼠肠系膜上动脉对去甲肾上腺素(noradrenine,NE)的收缩反应性降低,三羟异黄酮可在一定的剂量范围内明显改善失血性休克引起的血管低反应性;钒酸钠则可引起休克血管收缩反应性的进一步降低,且该作用可被0。
3)  vasohyporesponsiveness
血管低反应性
1.
Effects of disturbance of calcium homeostasis of smooth muscle cell on vasohyporesponsiveness in sepsis/septic shock;
平滑肌细胞内钙稳态变化在脓毒症/脓毒性休克血管低反应性形成中的作用
4)  Vascular hyporesponse
血管低反应
1.
Vascular hyporesponse or vasodilation is one of the key problems in treatment of these patents.
这也是以往认为的 ,在这些病理条件下 ,出现心血管低反应性的主要原因之一。
5)  Vascular hypo-responsive
血管收缩低反应性
6)  vascular reactivity
血管反应性
1.
Changes and organ diversity of vascular reactivity following endotoxic shock in rabbits;
内毒素休克后家兔血管反应性的变化规律及器官差异
2.
Objective To observe the effect of AVP on vascular reactivity and calcium sensitivity following hemorrhagic shock and its relations to V_(1a) and V_2 receptor in rats.
目的探讨精氨酸血管加压素(AVP)在恢复失血性休克动物血管反应性和钙敏感性中的可能机制和血管加压素V1 a、V2受体对其的作用。
3.
Objective To explore the changes of vascular reactivity following endotoxic shock and its relation with hemodynamic changes.
目的了解内毒素休克后血管反应性的变化规律及其与血流动力学变化的关系。
补充资料:超敏反应性血管炎


超敏反应性血管炎


由于超敏反应导致的血管壁的炎症病变。往往伴发肾脏、心脏以及皮肤的病变。
说明:补充资料仅用于学习参考,请勿用于其它任何用途。
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