1) nitric oxide synthase
NOS
1.
Objective To investigate the relationship between nitric oxide synthase (NOS) activity and schizophrenia.
目的通过测定首发精神分裂症患者和正常人的血清一氧化氮合酶 (NOS)活性 ,比较和分析他们之间的差异 ,探讨 NOS活性以及一氧化氮 (NO)能神经系统的功能变化与精神分裂症的关系。
2.
After 24 hours once exhaustive exercise the concentrations of nitric oxide in kidney,serum urea nitrogen,serum creatinine,urinary protein and nitric oxide synthase activity are determined.
一次性跑台力竭运动后24 h取材,检测肾组织NO含量、NOS活性和血清尿素氮、血清肌酐、尿蛋白含量,透射电镜观察肾细胞凋亡等病理变化。
3.
After the success of the models,divided them into 5 groups according to whether or not pre-given L-NAME(non-selective nitric oxide synthase inhibitor) and the doses: the A group,pre-given saline;the B1 group,pre-given L-NAME5mg;the B2 group, pre-given L-NAME10mg;the B3 group,pre-given L-NAME20mg;the B4 group, pre-given L-NAME30mg.
β-肾上腺素能受体激动剂能够舒张输尿管平滑肌,并激活NOS,增加输尿管组织释放NO剂量,其降低梗阻后的输尿管内压力作用部分是由NO介导的。
2) Nos terminator
Nos终止子
1.
Objective To establish the screening method of detecting genetically modified food in food stuffs by PCR amplifying the camv 35?S pr omoter and nos terminator.
方法 用改良溴化十六烷三甲基铵 (CTAB)法制备转基因抗除草剂〔大豆RoundUpReadyTMSoybean(RRS)〕和转基因抗虫玉米系列标准品Bt176Maximaizer的DNA ,PCR检测其内参照基因及camv 35S启动子和nos终止子。
2.
Methods The presence of GMO were investigated by PCR detection of camv 35S promoter and nos terminator, and the presence of RoundUp Ready TM Soybean (RRS), Bt176 Maximaizer or Mon810 YieldGard in GMO-positive samples were further determined by PCR detecting their specific DNA fragments respectively.
方法通过PCR检测camv35S启动子和nos终止子,筛选食品中是否含转基因成分,并进一步通过PCR检测RoundUpReadyTMSoybean(RRS)、Bt176Maximaizer和Mon810YieldGard的特异DNA片段,判断是否含这三种成分。
3.
The fragment of NOS terminator was obtained through the operating of DNA isolation and polymerase chain reaction(PCR)amplification from genetically modified tobacco.
以转基因烟草为材料,通过DNA提取、PCR扩增获得NOS终止子片段产物,以重叠延伸扩增法制备该片段的竞争性模板,并进行酶切验证。
3) NOS inhibitor
NOS抑制剂
1.
The experimental research of NOS inhibitor effect on the survival rates of rats with traumatic shock;
NOS抑制剂对创伤性休克作用的实验研究
2.
NOS inhibitor showed a protective effect,which could also inhibit the increase of the NO of PC12 cells.
[结论]DA对PC12细胞具有毒性作用,可以降低细胞的存活率,诱导细胞NO生成增加;NOS抑制剂具有一定保护作用,可明显抑制DA诱导PC12细胞NO生成增加。
4) NOS-NO signaling
NOS-NO通路
5) NOS activity
NOS活性
1.
Serumal NOS activity, NO content and liver tissue s IκBmRNA expression were assayed 4 hours after "two-hits".
5mg·kg-1),4h后检测血清NOS活性、NO含量和肝脏IκBmRNA的表达。
2.
In this paper, NOS activity and level of NOS is measured with exhaustive exercise model.
通过力竭运动模型 ,分别在运动后即刻、运动后 2h、运动后 12h和运动后 2 4h测定小脑皮层组织中NOS活性和MDA含量水平 ,结果发现运动后 2 4hMDA含量水平都没有显著的变化。
6) NO/NOS system
NO/NOS系统
1.
Research progress in interactions between NO/NOS system and opioid system;
NO/NOS系统与阿片系统相互作用的研究进展
补充资料：(R)-(-)-Glycidyl nosylate
分子式：C9H9NO6S
分子量：259.23
CAS号：115314-17-5
性质：熔点59-63°C。比旋光度-21.5° (c=2, CHCl3)。
分子量：259.23
CAS号：115314-17-5
性质：熔点59-63°C。比旋光度-21.5° (c=2, CHCl3)。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条