1) Nitrodeacetylation
硝化脱乙酰基
2) Deacetylation
[diə,siti'leiʃən]
脱乙酰基
1.
Effect of Deacetylation in Course of Konjac Glucomannan Carboxy Methylation and Structure Characterization of the Products;
脱乙酰基对魔芋葡甘聚糖羧甲基改性的影响及产物结构表征
2.
Abstract: Some methods of characterization and determination of transiormation ratio irorn acetylamine to amine in the deacetylation of chiun have been reviewed and evaluated.
本文对甲壳素脱乙酰基制取壳聚糖的反应中从乙酰胺基转变成胺基的转化率的各种表征方式和测定方法进行了详细综述和评价。
3.
The condition of deacetylation is studied,2-acetyl-4-chlorogalacose is obtained in dilute hydrochloric acid,and all deacetylated products are obtained in sodium hydroxide.
以α-D-三乙酰基-1-溴-4-氯半乳糖为原料,经Koenigs-Knorr反应、酯化反应,得到3-O-β-D-三乙酰基-4’-氯半乳糖熊果酸乙酯(UA2)、3-羟基-28-O-β-D-三乙酰基-4"-氯半乳糖熊果酸酯(UA5)和3-O-β-D-三乙酰基-4’-氯半乳糖熊果酸-28-O-β-D-三乙酰基-4"-氯半乳糖酯(UA8);研究了熊果酸衍生物脱乙酰基的条件,实验表明,在稀盐酸条件下得到4-氯半乳糖的2位保留乙酰基的产物,在固体氢氧化钠条件下得到全脱乙酰基的产物;共合成了9个熊果酸的4-氯半乳糖衍生物,所有目标化合物均为新化合物,其结构经NMR得以确认。
3) deacetylation
[diə,siti'leiʃən]
脱乙酰化
1.
Comment on deacetylation kinetics of chitin;
甲壳素脱乙酰化反应动力学规律评述
2.
Deacetylation Kinetics of Chitin and Application of Chitosan on Antibacterial Activity and Dyeing;
甲壳素脱乙酰化动力学及壳聚糖在抗菌染色中的应用研究
3.
Firstly,calcium carbonate is removed by marinating in hydrochloric acid;secondly,protein is removed by base and then chitin is obtained;then chitin reacts with concentrated base in 50 ℃ and through control the reaction time,chitosan are gained with different degrees of deacetylation(DD),respectively 85%,93%,99%.
利用H2O2的强氧化性制备低分子量分布的壳聚糖是将虾皮用HC l浸泡去除碳酸钙盐;再用稀碱除去蛋白质得甲壳素;然后浓碱在50℃与其反应,并控制反应时间,分别制备出脱乙酰度为85%,93%,99%的壳聚糖,最后用H2O2氧化不同脱乙酰化壳聚糖,得到不同低分子量的壳聚糖。
4) degree of deacetylation
脱乙酰化度
1.
In such reaction, the degree of deacetylation, the viscosity and infrared absorbance spectrum of deacetylation product are controlled by a variety of factors, such as the alkaline concentration , the temperature and time of reaction.
甲壳素经一次反应的脱乙酰化度不超过90％。
5) Histone deacetylase
脱乙酰化酶
1.
In t(8;21) AML, a stable association of AML1/ETO fusion protein with the nuclear histone deacetylase complex (HDAC) is crucial to repressing transcription of AMLl target genes and blocking differentiation of hematopoietic precursors.
目的：急性白血病M2b型(AML-M2b)由染色体易位产生的AML1-ETO融合蛋白异常募集组蛋白脱乙酰化酶(HDAC)从而抑制AML1靶基因的转录是关键的致病机制，并且DNA甲基化和组蛋白脱乙酰化两个重要的转录抑制机制内部存在着十分密切的关联。
6) ENU
乙酰基亚硝基脲
1.
Preliminary Study of Mice Mutation Induced by ENU;
乙酰基亚硝基脲诱导小鼠突变的初步研究
补充资料：1-氯甲酰基-3-乙酰基环亚乙基脲
分子式：C6H7ClN2O3
分子量：190.58
CAS号：41730-71-6
性质：密度1.528。熔点102-104°C。沸点278°C。闪点122°C。
分子量：190.58
CAS号：41730-71-6
性质：密度1.528。熔点102-104°C。沸点278°C。闪点122°C。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条