1) Newton-Raphson-Конτрович(NRK)
NRK法
2) NRK-52E cells
NRK-52E细胞
1.
Methods Divide NRK-52E cells into one control and six test groups.
目的探讨尾加压素Ⅱ(UⅡ)对大鼠近端肾小管上皮NRK-52E细胞增殖和细胞周期的影响及其机制。
2.
The mRNA expressions of MCP-1 and Col-Ⅰ in cultured NRK-52E cells were measured by semi-quantitive reverse transcriptase polymerase chain reaction(RT-PCR).
结果:NRK-52E细胞在TGF-β1刺激后MCP-1mRNA显著上调,在2h开始升高,8h达高峰,呈时间依赖性;PAI-1mRNA和蛋白的表达也显著增加;同时,TGF-β1还显著上调Col-ⅠmRNA的表达,24h为正常的2。
3) NRK cells
NRK细胞
1.
Objective To investigate the effects of cadmium chloride on cytotoxicity and lipid-peroxidation reaction in NRK cells.
方法CdCl2对NRK细胞染毒后,用MTT方法测定细胞的生长抑制,并测定细胞培养液中乳酸脱氢酶(LDH)、丙二醛(MDA)和超氧化物歧化酶(SOD)含量,观察镉的细胞毒性和对脂质过氧化影响。
2.
Objective To study early apoptosis and the expression of bax induced by cadmium chloride(CdCl_2) in NRK cells in vitro.
方法选择不同浓度(0~60μm ol/L)CdC l2体外作用NRK细胞12 h及20μm ol/L CdC l2体外作用NRK细胞不同时间(10 m in~24 h),用流式细胞仪检测细胞凋亡;用逆转录-多聚合酶链扩增反应(RT-PCR)的方法观察20μm ol/L CdC l2作用NRK细胞不同时间(0。
3.
Objective To explore the effects of MAPK inhibitors on mRNA expression of apoptosis genes Bcl-2,Bax and Caspase-3 in NRK cells treated with cadmium chloride.
5,5,10μmol/L)染毒大鼠NRK细胞12h,p38MAPK抑制剂SB20358010μmol/L和ERK1/2抑制剂PD9805910μmol/L预处理NRK细胞0。
4) NRK
NRK方式
5) NRK-52EA
NRK-52EA细胞
1.
Objective To investigate the effects of fluctuating high glucose on the transdifferentiation of tubule endothelial cell line NRK-52EA in mouse.
结论:间歇性高糖能通过氧化应激诱导肾小管导管上皮细胞表达TGF-β1、MMP2,并促进NRK-52EA细胞转分化为α-SMA细胞。
6) AS method
AS法
1.
This paper determines the ideal environmental capacity by using the A value method,distributes the environmental resource by using the AP and AS methods,which achieves the standards of both the concentration and the total amount.
通过A值法核定理想环境容量,采用AP,AS法对环境资源进行分配,从而达到“浓度与总量双达标”。
补充资料：法性属法为法性土
【法性属法为法性土】
谓真如法性之理，譬如虚空，遍一切处，乃是法身所证之体，即为所依之土，故名法性属法，为法性土。
谓真如法性之理，譬如虚空，遍一切处，乃是法身所证之体，即为所依之土，故名法性属法，为法性土。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条