1) diarrhetic shellfish poisoning
腹泻性贝毒
1.
Analysis of paralytic shellfish poisoning and diarrhetic shellfish poisoning of bivalves in seafood market of Guangzhou;
广州市售贝类麻痹性贝毒和腹泻性贝毒污染状况分析
2.
The determination of diarrhetic shellfish poisoning of sea shellfish was done in China coast by HPLC in 2001.
使用HPLC方法分析了2001年8～10月间采集的我国沿海双壳贝类体中的腹泻性贝毒(DiarrheticShellfishPoisoning,DSP)。
2) diarrhetic shellfish poison
腹泻性贝毒
1.
A method based on protein phosphatase enzyme activity inhibition for the detection of diarrhetic shellfish poison (DSP) was used to analyze the DSP toxicity in three oyster samples.
基于腹泻性贝毒(Diarrhetic Shellfish Poison,DSP)中大田软海绵酸(Okadaic acid,OA)和鳍藻毒素(Dinophysis toxins,DTXs)能够抑制蛋白磷酸酶活力的特点,人们建立了一种利用碱性蛋白磷酸酶活力变化检测贝类中大田软海绵酸毒性当量的生物化学测试方法。
2.
To explore the nutrient properties of Prorocentrum lima and biosynthesis mechanism of diarrhetic shellfish poison (DSP), the growth and activities of alkaline phosphatase of Prorocentrum lima were observed under different phosphorus sources.
探讨了不同磷源条件下利玛原甲藻的生长情况,分析了碱性磷酸酶在营养盐利用方面的作用,对不同营养盐条件下腹泻性贝毒(diarrhetic shellfish poison,DSP)的合成情况进行了比较和分析。
3) DSP
腹泻性贝毒
1.
Prorocentrum lima is a red-tide organism that is responsible for diarrhetic shellfish poisoning(DSP).
采用单因子实验,设置4个浓度梯度:12μmo·lL-1、25μmol·L-1、50μmo·lL-1和100μmol·L-1,研究了NaNO3、NH4Cl和尿素对利玛原甲藻(Prorocentrum lima)的生长、硝酸还原酶(nitrate reductase,NR)活性以及腹泻性贝毒(diarrhetic shellfish poisoning,DSP)产生的影响,对比分析了利玛原甲藻对3种不同氮源的利用特征。
2.
Objective:To provied information for the HABs toxin synthesis mechanism and explore the role of polyketide synthase in the synthesis of diarrheic shellfish poisoning(DSP) toxins,potential polyketide synthase(PKS) genes in Prorocentrum lima was amplified,the homology analysis of the PKS gene in related species was conducted and phylogenetic tree was constructed.
目的:获得利玛原甲藻(Prorocentrum lima)聚酮合成酶(polyketide synthase,PKS)基因,进行序列分析和同源性比较;观察不同生长期、不同营养盐条件下利玛原甲藻的产毒情况及PKS基因的表达变化,分析PKS基因与藻毒素合成的关系,揭示PKS在腹泻性贝毒(diarrheic shellfish poisoning,DSP)合成中的作用。
4) Diarrheic shellfish poisoning
腹泻性贝毒
1.
In order to explore the roles of HSP70 gene in resistant mechanism of shellfish to diarrheic shellfish poisoning(DSP)toxins,the accumulation and cleaning action of DSP toxins in Perna viridis were studied,and HSP70 gene expression induced by DSP toxins were detected,too.
以利玛原甲藻(Prorocentrum lima)为饵料,通过室内滤食实验,考查了腹泻性贝毒(diarrheic shellfishpoisoning,DSP)在翡翠贻贝(Perna viridis)体内的累积与排出规律,分析了HSP70基因的表达与毒素累积与排出之间的关系。
5) diarrhetic shellfish poisoning
腹泻性贝毒素
1.
The diarrhetic shellfish poisoning(DSP) toxicity in shellfish was determined with mouse bioassay later years inshore in the South China Sea.
通过对近年来南海近岸海域贝类样品的腹泻性贝毒素小白鼠生物法检测结果的分析,表明广西主要的染毒海域是涠洲赤控区、铁山港及防城港等地,海南主要的染毒海域是陵水赤控区、海口、莺歌海、三亚及洋浦等地,广东主要的染毒海域是大鹏湾赤控区、大亚湾及北津港等地;广西的涠洲赤控区、海南的陵水赤控区及广东的大鹏湾赤控区DSP毒素含量值较高;在染毒的贝类种类方面,广西和海南比较有普遍性,广东主要以华贵栉孔扇贝和翡翠贻贝为主。
2.
In order to improve the mouse bioassay for diarrhetic shellfish poisoning detection,different concentrations of diarrhetic shellfish poisoning(DSP)—Okadaic acid(OA)were injected into Kunming strain mice by intraperitoneal route,and the toxic symptom was observed to determine the dosage for one mouse unit(MU)and develop the curve of dosage versus death time.
为完善腹泻性贝毒素软海绵酸(Okadaic acid,OA)的小鼠生物学检测技术,以昆明系小鼠为实验材料,应用小鼠生物学检测法检测了OA的毒性。
6) diarrhetic shellfish poison
腹泻性贝类毒素
1.
The preliminary study of diarrhetic shellfish poison in Zhoushan fishing ground and its adjacent area;
舟山渔场及其邻近海域腹泻性贝类毒素的初步研究
2.
The investigation of diarrhetic shellfish poison(DSP) in oyster from 23 major culture waters in South China Sea was carried out in 2006 and 2007.
对2006年和2007年南海海域23个重要养殖水域牡蛎体中腹泻性贝类毒素(DSP)进行了调查,结果表明:甲子港、唐家湾、镇海湾、安埔港、防城港、八所港和榆林港等7个水域牡蛎体DSP毒性呈阳性结果,占调查水域的30。
补充资料：腹泻啶
CAS：53179-11-6
分子式：C29H33ClN2O2
分子质量：477.04
中文名称：盐酸洛哌丁胺；咯哌丁胺；4-对氯苯-4-羟基-N,N-二甲基-α,α-二苯-1-对哌啶基丁酰胺；氯苯哌丁胺；苯丁哌胺；腹泻啶
英文名称：4-(4-chlorophenyl)-n,n-dimethyl-alpha,alpha-diphenyl-4-hydroxy-1-piperid ineb；utanamide；loperamide；4-(4-Chlorophenyl)-4-hydroxy-N,N-dimethyl-,-diphenyl-1-p-piperidinebutanamide
性状描述：该品常用其盐酸盐（C29H33CIN2O2·HCL[34552-83-5]）；哌丁胺盐酸为白色至微黄色结晶性粉末，熔点222-223℃。易溶于甲醇；氯仿；冰醋酸，溶于乙醇；DMF，难溶于水；异丙醇；丙酮，不溶于乙醚。
生产方法：以二苯乙腈为原料，在氨基钠存在下与环氧乙烷反应后用盐酸水解，再在室温下用48%HBr-HAc开环，经氯化；二甲胺化后与4-对氯苯基-4-羟基哌啶在甲基异丁基酮及碳酸钠存在下缩合制得洛哌丁胺。以二苯乙腈计，总收率13.4%。
用途：洛哌丁胺为苯基哌啶类止泻药，具有长效作用，用于治疗功能性腹泻以及因胃肠切除或肠器质性损害等引起腹泻，疗效甚佳。小鼠口服LD50为105mg/kg，大鼠（7d）为185mg/kg。
分子式：C29H33ClN2O2
分子质量：477.04
中文名称：盐酸洛哌丁胺；咯哌丁胺；4-对氯苯-4-羟基-N,N-二甲基-α,α-二苯-1-对哌啶基丁酰胺；氯苯哌丁胺；苯丁哌胺；腹泻啶
英文名称：4-(4-chlorophenyl)-n,n-dimethyl-alpha,alpha-diphenyl-4-hydroxy-1-piperid ineb；utanamide；loperamide；4-(4-Chlorophenyl)-4-hydroxy-N,N-dimethyl-,-diphenyl-1-p-piperidinebutanamide
性状描述：该品常用其盐酸盐（C29H33CIN2O2·HCL[34552-83-5]）；哌丁胺盐酸为白色至微黄色结晶性粉末，熔点222-223℃。易溶于甲醇；氯仿；冰醋酸，溶于乙醇；DMF，难溶于水；异丙醇；丙酮，不溶于乙醚。
生产方法：以二苯乙腈为原料，在氨基钠存在下与环氧乙烷反应后用盐酸水解，再在室温下用48%HBr-HAc开环，经氯化；二甲胺化后与4-对氯苯基-4-羟基哌啶在甲基异丁基酮及碳酸钠存在下缩合制得洛哌丁胺。以二苯乙腈计，总收率13.4%。
用途：洛哌丁胺为苯基哌啶类止泻药，具有长效作用，用于治疗功能性腹泻以及因胃肠切除或肠器质性损害等引起腹泻，疗效甚佳。小鼠口服LD50为105mg/kg，大鼠（7d）为185mg/kg。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条