1) Arsenic poisoning
砷中毒
1.
Change of CD44 adhesion molecule in the peripheral blood lymphocytes of patients with coal arsenic poisoning and its clinical significance;
燃煤砷中毒患者外周血淋巴细胞CD44黏附分子的变化及临床意义
2.
The influence of Pine Pollen on rDNA transcription activity of peripheral blood lymphocyte in arsenic poisoning rats;
松花粉对砷中毒大鼠外周血T淋巴细胞rDNA转录活性的影响
3.
The protective effect of Pine Pollen on arsenic poisoning rats;
松花粉对砷中毒大鼠的防护作用
2) Arsenism
['ɑ:sinizəm]
砷中毒
1.
Investigation on Arsenic concentration in drinking water and disease state of Arsenism in Shanyin County;
山阴县饮水砷含量及砷中毒病情调查
2.
Study on change of serum enzymes activity in chronic arsenism rats;
慢性砷中毒大鼠肝脏血清酶活性变化分析
3.
A study on plasma cytokines of patients with endemic arsenism after 15 years intervention experiment;
改水15年后地方性砷中毒患者细胞因子水平检测分析
3) arseniasis
[ɑ:si'naiəsis]
砷中毒
1.
A Survey on Endemic Arseniasis in Zhejiang Province;
浙江省地方性砷中毒调查
2.
To account the drinking-water type of arseniasis in research area, three method-opening a well for recreational water, withdrawal water from side channel and physical-chemical method were adopted for arsenic removal-recreational water.
本文针对研究区饮水型砷中毒，采用了打井改水、傍渠取水和理化除砷改水等三种方法，取得了较为满意的结果。
3.
MATERIALS AND METHODS: Using telomeric repeat amplification protocol(TRAP) assay and ABC immunohistochemical technique, the level of telomerase activation and PCNA expression were evaluated in 29 skin lesions of arseniasis patients and 10 cases of normal skin.
材料与方法:采用端粒重复序列扩增法(TRAP)和免疫组化ABC法检测29例砷中毒患者病损皮肤及10例正常皮肤组织中的端粒酶活性和PCNA表达情况。
4) arseniasis and fluorosis
砷氟中毒
1.
But there are some serious eco-geochemical problems including soil salification,which restricts regional agricultural development,and endemic diseases of arseniasis and fluorosis,which badly affect the human health.
作为我国北方重要的粮食生产基地可以为全国生产出优质的绿色产品;但河套地区也具有比较严重的生态地球化学问题,首先是土壤的盐渍化制约了河套地区农业的发展,其次是全区以砷氟中毒为主的地方病比较严重,极大地影响了该区人民的身体健康。
5) fluorine and arsenic poisoning
氟、砷中毒
6) acute arsenic poisoning
急性砷中毒
1.
Impact of acute arsenic poisoning on human eyes;
急性砷中毒对眼部的损害
补充资料：砷中毒
| 砷中毒 arsenic poisoning 由砷的氧化物、盐类及有机化合物引起的人体损害。特别是三氧化二砷（As2O3，俗称信石或砒霜）毒性最大。农村常用此拌种、杀灭农业昆虫和灭鼠，中毒机会比较多，也有因误服而中毒的；在接触砷化合物的生产中和临床使用含砷的外用药时也可发生砷中毒。砷及其化合物通过呼吸道、消化道或皮肤进入体内。食入引起的急性中毒表现急性胃肠炎症状，有血便。重者有脱水、休克、抽搐、谵妄、昏迷，也可损害心肌。1～3周后可出现多发性末梢神经炎和神经根炎，表现为四肢无力、麻木、麻痹，感觉迟钝或消失，呈手套袜套样分布，肌腱反射减低或消失，肌肉萎缩。也可有中毒性肝损害、出血倾向、皮肤黑变；指甲出现白色横纹。吸入中毒时除上述症状外还可有呼吸系统症状。慢性中毒有神经衰弱综合征、消化道紊乱、肝损害、末梢神经炎和皮肤损害等表现。长期接触砷及砷化合物者，皮肤癌、肝癌和肺癌的发病率高于正常人。根据接触史、临床表现和尿、呕吐物、毛发、指甲等砷的含量增高可诊断。 砷中毒的特效解毒剂为二巯基丙醇磺酸钠或二巯基丙醇，可肌肉注射。此外还可采用对症和支持疗法。 |
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条